a reply to:
sapien82
That’s pretty interesting about ECS being a defensive mechanism. Never thought of it that way. But I’ve contemplated it a lot in past, and think
there’s some kind of co-evolution with cannabis, for us to have that receptor network within us. What are your thoughts?
I haven’t been able to stop thinking about this though, which could be our suppressed defense mechanism of sorts. In the 4chan link above (I know,
but have to go where the info is), if you follow their link to the previous page (Vol II), you’ll find the text I’m about to share. It’s been
blowing my mind, quite honestly. I really think they’re onto something. Also very interesting to me, how they’re using ‘The Great Filter’ in
the thread titles.
Basically, this researcher is sharing his findings, on how he believes the organism is deliberately concentrating our endogenous '___', into our
pineal glands. This is a manipulation of our biology, because endogenous '___' is supposed to be spread throughout the body. So, once we clear the
parasite, '___' will naturally begin to form throughout the body, as it should. Fascinating stuff:
Author – Peribsen
boards.4channel.org...
“(1)
One of the larger chunks of time I’ve spent lately has been on familiarizing myself with neurotransmitters, neural stem cells and differentiation,
and what a body free of disease might be able to accomplish re: healing itself and maintaining itself in those areas. Main idea being to test and
biohack to a degree that is removing pathogenic activity as much as possible without damaging myself along the way. No one likes to hurt themselves if
they’re aiming for healing old damage/closing immune issues out. It’s like debugging but for your own code and wiring.
We’ve all heard of dimethyltryptamine at some point. We’re told that it’s usually only generated (or mainly) in the pineal gland. This is false.
I’m going to show you the math on endogenously generated dimethyltryptamine being a state the body should be able to maintain, at the least for
on-the-moment use throughout the body. I’ll start with the basics of neurochemistry as related to dimethyltryptamine ('___' – mods, sorry I’m
only talking synthesized withing the body naturally here).
www.ncbi.nlm.nih.gov... - N, N-Dimethyltryptamine ('___'), an Endogenous Hallucinogen
You obtain tryptophan from a few sources that are foods or supplements. Personally that’s the only recommendation I would make in reference to this
particular hack that I’m referring to. Your body can do it with food sources though in this case. Tofu and fish are highest that I know of with
eggs, milk, and cheese following iirc. So now you have tryptophan in the body and your body wants to turn that into tryptamine via decarboxylation –
tryptamine is a base component of most neurotransmitters further down the line.
(2)
>In addition to INMT, production of '___' requires aromatic-L-amino acid decarboxylase (AADC), which removes the carboxyl group from dietary
tryptophan to form tryptamine, the essential '___' precursor that can be rapidly metabolized by monoamine oxidase.
www.ncbi.nlm.nih.gov... - Biosynthesis and Extracellular Concentrations of N,N-dimethyltryptamine ('___') in Mammalian
Brain
Two things to note from that article. One is that the INMT expression (which leads to '___') for human and rat is shown in the brain, adrenal, kidney,
lung, heart, and visual cortex. Also note that tryptamine can be metabolized by a term you may recognize – monoamine oxidase, or MAOs. A marker that
is overexpressed in many brain chemistry disorders. This leads to the next point we need to consider related to the overall place that cryptococcus
infection in the body affects our chemistries.
>Neurotransmitters such as dopamine in brain tissue are contained within vesicles and degraded by monoamine oxidase, an enzyme found in glial
cells.
www.ncbi.nlm.nih.gov... - Cryptococcus neoformansmelanization incorporates multiple catecholamines to produce polytypic
melanin
And this article lends further support, look at the information in figure 1.
>Polymorphisms in these enzymes, the receptors their products bind to (e.g., 5-HT2A), and/or the metabolic enzymes that degrade their products (e.g.,
MAO) could affect the levels and effects of the endogenous '___'s through multiple permutations of SNPs as discussed throughout the manuscript.
www.ncbi.nlm.nih.gov... - Indolethylamine-N-methyltransferase Polymorphisms: Genetic and Biochemical Approaches for
Study of EndogenousN,N,-dimethyltryptamine
(3)
As seen in the article below, glial cells such as microglia (the immune cells of the brain) are capable of generating the monoamine oxidases that
degrade tryptamine stores. Lowered tryptamine stores means less endogenously produced neurotransmitters and neurohormones. And '___' with its base
chemistry expressed throughout the body is now being considered as a primary neurotransmitter. Stop the cryptococcal oxidation via glial cells,
produce your stores of dimethyltryptamine naturally.
www.ncbi.nlm.nih.gov... - Role of microglia in fungal infections of the central nervous system
This math shows an additional layer of human biology that cryptococcus neoformans is manipulating. At the level of expression that we’ve looked at,
dimethyltryptamine should be present widely throughout the body. It shows up in nearly all nervous system tissues and several major organs. It’s a
chemical closely associated with transcendent states, primarily meditation and prayer. Certain other states like orgasm, death, NDEs, and OBEs are
also tied to this neurotransmitter. Two key notes from the following article should be held in mind.
>Perinatal INMT ActivityLevels of INMT in the placenta are higher than in adults. It is speculated that activity in fetal lungs compensates for
difference. INMT activity in rabbit lung is relatively high in fetus, increases rapidly after birth and peaks at 15 days of age. It then declines to
mature levels and remains constant through life.
>This model predicts that the sensory-altering effects of administered psychedelics are a result of the compound acting directly via
neuropharmacological mechanisms in regions of the CNS involved in sensory perception. More simply, '___' may potentially act as a neurotransmitter to
exert a signaling function in regions of the CNS, which are involved in sensory perception.
www.ncbi.nlm.nih.gov... - Neuropharmacology of N,N-Dimethyltryptamine
(4)
From this information it is reasonable to assume that the MAO activity against tryptamine begins shortly after birth, and then tapers off to
“mature” levels that primary are found in the pineal gland/hippocampus. It is also indicated that '___' is a neurotransmitter, production of which
is being interrupted by cryptococcus via these MAOs."