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I think the phospholipids in the lecithin are really noticeably good for me, totally aside from the ascorbic. Too bad they taste so utterly vile.
Julie Washington
What I found most interesting in the study is that oxygen degrades Vitamin quicker than anything. Oxygen is worse that heat.
So keep your Vitamin C in the Fridge sealed tight!
Timely
I hope you are not a new " player " trying to shut down this thread.
Conspiracy theories abound !
Have you tried the protocol - personally ?
Timely
Good to hear of your positive experience with this.
Personally I have not experienced the 'euphoric' effect, yet I have not taken I'll either.
RedCairo
Wow. Seriously, I want what you're having. ;-)
I feel significantly better when taking fairly big doses regularly but not really per-dose and not euphoric, just 'better.' I wonder how much effect the lecithin has versus the ascorbic for many. Guess there's no way to tease that factor apart in this case.
"For example, it is well established that ascorbate is essential for catecholamine biosynthesis in neural tissues, serving as a co-factor for dopamine..."
The highest concentrations of ascorbate in the body are found in the brain and neuroendocrine tissues such as adrenal...
...Ascorbate is proposed as a neuromodulator of glutamatergic, dopaminergic, cholinergic and GABAergic transmission and related behaviors. Neurodegenerative diseases typically involve high levels of oxidative stress and thus ascorbate has been posited to have potential therapeutic roles against ischemic stroke, Alzheimer's disease, Parkinson's disease and Huntingdon's disease.
Intraperitoneal ascorbate (125 mg/kg) reversed memory deficits in mice induced both by age and scopolamine treatment in a transfer latency task in the elevated plus maze and a passive avoidance task and in a habituation-based task in a light-dark paradigm. When ascorbate was given orally for 30 days (300mg/kg) in conjunction with vitamin E in aged mice (15 months), it also improved performance on a passive avoidance task. Improvement was not noted in 3 month old mice or when ascorbate was administered alone in that study. It should be noted, however, that these are atypical tests of learning and memory. The first two are variants of tasks more often used to measure anxiety than learning, and the third relies on learned associations between a response and an electric shock
Ascorbate provided in drinking water has been shown to reduce the fear response in Japanese quail chicks tested in a less stressful light-dark emergence paradigm.
After 2 weeks of vitamin E deprivation, plasma and brain α-tocopherol concentrations were decreased by 65% and 32%, respectively. These animals gained weight and appeared completely normal. However, within 5-6 days of removing vitamin C from their diet, most of the animals developed a progressive ascending paralysis and died within 24 hours. No neurologic signs were apparent in animals with single deficiencies of vitamins C or E.
That ascorbate is important for neuronal maturation and function, as well as for protection of the brain against oxidant stress is well supported by the evidence presented in this review.
The vitamin is maintained at high concentrations in brain and in neurons in particular relative to other organs. In addition, strong homeostatic mechanisms maintain brain and neuronal ascorbate concentrations within very tight limits. Thus, not only is it difficult to deplete brain ascorbate, but it is also difficult if not impossible to increase levels for more than a short period above those set by uptake and recycling mechanisms. Whereas oral supplements generally increase brain ascorbate by only 20% at most, larger relative increases may occur if significant oxidant stress has caused localized ascorbate deficiency in brain areas affected by neurodegeneration or inflammation.
Study of the role of ascorbate in human brain function has been limited, but with the availability of suitable mouse models, ascorbate deficiency or excess can be studied in more detail, particularly with regard to effects of the vitamin on brain development, neurotransmitter function and responses to inflammatory or oxidant stresses, such as might exist in cerebral atherosclerosis or in several neurodegenerative diseases.
Julie Washington
reply to post by Tarasco
Tarasco,
That is an interesting dilemma. I also buy Swanson's products.
I haven't ordered recently. My latch batch of non gmo soy grandules was ordered several months ago. Same 3 lb container and the quality appear good, a nice golden color.
I will be interested in hearing what you find out.
toastyr
reply to post by Tarasco
Interesting, what were your symptoms? I have been using the swansons sunflower lecithin for a while now. On my 5th lb. and they've all looked the same. I ordered twice, three lbs. each time.
papajake
reply to post by Tarasco
I haven't tried the powder form of Sunflower Lecithin. I've been using the gooey liquid stuff from Azure Standard. It's worked great for me and it's non-gmo. I'm on my second 1 gallon bottle and getting ready to order another. They've been consistent in texture and color. It seems to take a bit longer to sonicate but I like it much better than soy.