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I posted about Gibb's paper back when it first came out. That's from back in May. Like I said, there were two strains he did not know were already in circulation in pigs (found in test samples form N Carolina, I believe). It was this missing data which led him to his conclusion. Send him an email and see where he stands now, he pulled his paper from publication in the journals once the information came out about the missing strains.
At the time, I supported his claims also because my private sources were also coming to that conclusion. If you want more detail on the missing strain issue let me know and I'll ask about it.
# Since 1997, novel viruses of three different subtypes and five different genotypes have emerged as agents of influenza among pigs in North America. The appearance of these viruses is remarkable because there were no substantial changes in the overall epidemiology of swine influenza in the United States and Canada for over 60 years prior to this time. Viruses of the classical H1N1 lineage were virtually the exclusive cause of swine influenza only from the time of their initial isolation in 1930 through 1998. [Virus Research 2002 May 10;85(2):199–210]
# Since 1998 triple reassortant swine influenza viruses have been isolated from pigs in the United States and a human case of a triple reassortant swine flu infection was reported in the US as early as 2005. This patient was exposed to freshly-killed pigs. [Emerging Infectious Diseases 2008 Sep; 14(9):1470–2]
# A report published in The New England Journal of Medicine in June of 2009 indicates triple-reassortant swine flu influenza A H1 viral infections were reported among a few humans who worked closely with pigs from 2005 to 2009. [New England J Medicine. 2009 Jun 18; 360(25):2616–25] However, this is not the exact flu virus that is currently sweeping globe with a late season onset in 2009.
# Michael Gregor MD claims crowding in pig farms is a factor in the development of the current triple reassortant flu virus infecting humans, asserting its precursor was first discovered in pig herds in North Carolina in 1998. North Carolina is the home of the nation’s largest pig farms. And it’s true, 3/4ths of the genetic material in the H1N1 human swine flu of 2009 comes from this 1998 pool of swine viruses. [Humane Society, August 26, 2009] But again, this may be a misdirection.
# Sometimes the obvious is not recognized. With all of the prior discussion about swine-origin influenza, this H1N1 late-2009 flu season virus is not currently found in pig herds, says Vincent Racaniello PhD, Professor of Microbiology at Columbia University Medical Center. The virus may have originated in a human! Asking the question "where did ‘pig zero’ come from?" may be the wrong question. Wikipedia accurately acknowledges that "the 2009 H1N1 virus is not zoonotic swine flu (zoonotic = transmitted from animals to people), as it is not transmitted from pigs to humans, but from person to person."
# The tracking of the lineage of this virus has been perplexing if for no other reason than the misdirection to investigate herds of pigs. News reporters may have been misled from the outset. For example, Karen Kaplan of The Tribune Newspaper wrote on Sept. 13, 2009 that "Somehow, a single pig became simultaneously infected with that virus and a pure swine flu strain found in pigs in Europe and Asia. The two strains swapped genetic material to produce the new H1N1 strain, which then began to infect humans." But the virus has not been detected in swine herds so far. It is similar to other reassortant flu viruses, but not the same.
# Over-reliance upon vaccination and anti-flu drugs increases genetic pressure for flu viruses to mutate into treatment-resistant forms. Genetic mutation is accelerated with the use of vaccines and antivirals. [PLoS One 4(3):e4915, March 18, 2009; Virology Journal 5: 133–39, October 2008]
# Just how do flu viruses mutate within a human? It is known that bursts of free radicals can induce gene mutations in viruses. [FASEB Journal 2000 Jul; 14(10):1447–54] Vaccination of birds and humans against the flu has been shown to apply genetic pressure and induce more rapid genetic drifts. [Virology Journal 2008; 5: 15]
# Bursts of free radicals emanate from what is called a "cytokine storm." Cytokines are substances that are secreted by specific cells of the immune system and can increase 1000-fold during infection. Over-reactive cytokine response is attributed to the fatality rate of flu viruses. [Mikrobiyol Bulletin 2008 Apr;42(2):365–80]
# Whole inactivated or so-called "killed" viral vaccines, which are the most commonly used, are plagued by their inability to activate antibodies. So they generally require strong adjuvants and several injections to be fully effective. [Clinical Microbiology Review 2007; 20: 489–510] Adjuvants are pharmacological or immunological agents that modify the effect of other agents (e.g., drugs, vaccines).
# MF59, the favored squalene-based adjuvant employed in both swine herd and human vaccines, could possibly trigger mutations that make flu viruses treatment resistant. Researchers indicate MF59 does indeed induce greater immune response due to its ability to generate cytokines! [Proceedings National Academy Sciences U S A. 2008 Jul 29; 105(30):10501–6]
# It has been repeatedly shown that nutritional deficiencies, particularly of vitamin E and the trace mineral selenium, exacerbate the immune response to replicating flu viruses and that this then leads to alterations in the gene makeup of the viral genome, resulting in a mortal outcome. [Trends Microbiology 2004 Sep;12(9):417–23] Nutritional deficiency not only affects the host response to the virus but also the viral genome itself. [Proceedings Nutrition Society 1999 Aug;58(3):707–11] To limit mortality, public health officials should be fortifying foods with selenium and vitamin E to head off the possibility of gene mutations among flu sufferers. [J Trace Element Med Biol. 2007;21(1):52–62]
Originally posted by really
Well, I don't follow Jane's forum. Never have. This seems to be a fact. I really don't care what Jane has to say. Never listened to her to begin with.
Originally posted by ecoparity
Originally posted by really
Well, I don't follow Jane's forum. Never have. This seems to be a fact. I really don't care what Jane has to say. Never listened to her to begin with.
I keep reading that same statement from people who post about the Baxter incident, don't they realize Jane B is THE sole source behind the entire story?
Every element of the story going around that is factually wrong comes directly from her - that it was a vaccine that was contaminated, that 72 kilos of material were involved, that it was sent to 18 labs - all her assertions.
[edit on 21-11-2009 by ecoparity]
Feb. 24 (Bloomberg) -- Baxter International Inc. in Austria unintentionally contaminated samples with the bird flu virus that were used in laboratories in three neighboring countries, raising concern about the potential spread of the deadly disease.
The contamination was discovered when ferrets at a laboratory in the Czech Republic died after being inoculated with vaccine made from the samples early this month.
The material was intended for use in laboratories, and none of the lab workers have fallen ill.
Originally posted by squiz
I wasn't talking about any conspiracy. Did they or did they not release live bird flu, unlabeled, together with seasonal flu vaccine components?