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A way to reverse ageing has been discovered which allows withered muscle to rebuild itself by turning back a “biological clock”.
The effect has already been demonstrated on human muscle tissue in the laboratory.
Scientists in the US believe the breakthrough could lead to new treatments that rejuvenate and strengthen ageing bodies or combat degenerative diseases.
Originally posted by Republican08
This surprisingly upsets me....
Why you ask, well if we can do this....
Where the hell is my flying car!!!!
Originally posted by Wormwood Squirm
Can they make it focus entirely on the penis? What is the stock symbol of this company?
Originally posted by Wormwood Squirm
Can they make it focus entirely on the penis? What is the stock symbol of this company?
Originally posted by space cadet
This is wonderful, only what about the skin over the muscle? Muscle strenghthening is going to help you feel and look somewhat younger, but the skin is what really really shows the age of an individual.
Originally posted by InfaRedMan
reply to post by cosmicpixie
No doubt NASA will find this a useful application to combat muscle wastage in those subjected to long term space travel... like astronauts traveling to Mars and beyond. Now they just need to stop the bones from decalcifying.
IRM
Very little remains known about the regulation of human organ stem cells (in general, and during the aging process), and most previous data were collected in short-lived rodents. We examined whether stem cell aging in rodents could be extrapolated to genetically and environmentally variable humans. Our findings establish key evolutionarily conserved mechanisms of human stem cell aging. We find that satellite cells are maintained in aged human skeletal muscle, but fail to activate in response to muscle attrition, due to diminished activation of Notch compounded by elevated transforming growth factor beta (TGF-)/phospho Smad3 (pSmad3). Furthermore, this work reveals that mitogen-activated protein kinase (MAPK)/phosphate extracellular signal-regulated kinase (pERK) signalling declines in human muscle with age, and is important for activating Notch in human muscle stem cells. This molecular understanding, combined with data that human satellite cells remain intrinsically young, introduced novel therapeutic targets. Indeed, activation of MAPK/Notch restored youthful myogenic responses to satellite cells from 70-year-old humans, rendering them similar to cells from 20-year-old humans. These findings strongly suggest that aging of human muscle maintenance and repair can be reversed by youthful calibration of specific molecular pathways.