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Provide me evidence he wasn't murdered,....
.... because all the evidence suggests he was.
If the vaccine caused his death, then it's murder,.....
....because people were forced to take it under threat of losing their livelihood and ability to participate in normal society.
Even if Shane begged for the vaccine it doesn't change the situation, every single person who dies as a result of this vaccine is the victim of murder because they were conned.
Well there's a thing called an autopsy which could presumably prove he died from smoking or something else. If they can't prove anything either way then I can only make the most logical conclusion, which is that he was killed by a vaccine which has documented side effects which cause heart issues.
originally posted by: Freeborn
a reply to: ChaoticOrder
So, when someone dies the onus is on to prove they were not murdered?
When did that happen?
originally posted by: ChaoticOrder
a reply to: and14263
Seems very relevant to me, both in their early 50's, both fairly athletic and fit, both claimed to have heart failure due to drugs...
originally posted by: ChaoticOrder
Shane Warne... didn't do illegal drugs... I highly doubt he was a heavy smoker due to the career he had.
originally posted by: continuousThunder
originally posted by: ChaoticOrder
Shane Warne... didn't do illegal drugs... I highly doubt he was a heavy smoker due to the career he had.
Are we uhh.... talking about the same Shane Warne?
The three most advanced vaccines (from Oxford/AstraZeneca, Pfizer/BioNTech and Moderna) all work by getting our own cells to make copies of the virus spike protein. The Oxford vaccine achieves this by introducing the spike protein gene via a harmless adenovirus vector. The other two vaccines deliver the spike protein gene directly as mRNA wrapped in a nanoparticle. When our own cells make the spike protein, our immune response will recognize it as foreign and start making antibodies and T cells that specifically target it.
While the findings themselves aren't entirely a surprise, the paper provides clear confirmation and a detailed explanation of the mechanism through which the protein damages vascular cells for the first time.
There's been a growing consensus that SARS-CoV-2 affects the vascular system, but exactly how it did so was not understood. Similarly, scientists studying other coronaviruses have long suspected that the spike protein contributed to damaging vascular endothelial cells, but this is the first time the process has been documented.
In the new study, the researchers created a "pseudovirus" that was surrounded by SARS-CoV-2 classic crown of spike proteins, but did not contain any actual virus. Exposure to this pseudovirus resulted in damage to the lungs and arteries of an animal model--proving that the spike protein alone was enough to cause disease. Tissue samples showed inflammation in endothelial cells lining the pulmonary artery walls.
The team then replicated this process in the lab, exposing healthy endothelial cells (which line arteries) to the spike protein. They showed that the spike protein damaged the cells by binding ACE2. This binding disrupted ACE2's molecular signaling to mitochondria (organelles that generate energy for cells), causing the mitochondria to become damaged and fragmented.
Previous studies have shown a similar effect when cells were exposed to the SARS-CoV-2 virus, but this is the first study to show that the damage occurs when cells are exposed to the spike protein on its own.