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A group of European researchers led by Pagona Lagiou of the University of Athens Medical School in Greece assessed the diets of more than 43,000 Swedish women ages 30 to 49, and followed them for an average of almost 16 years. Women who consumed a diet consisting of low carbohydrate and high protein intake were at a 5 percent higher risk of cardiovascular disease later. By the end of the end of the study period, 1,270 women developed heart disease.
Consuming as little as 20 fewer grams of carbohydrates and 5 more grams of protein per day accounted for the increase, the researchers found.
However that does not prove that diabetes results from an Auto-immune deficiency. Organs do fail if they are overtasked and in this case that seems to more reasonable cause to such a condition.
All meats fall into the category of high fat as compared to a Vegan diet in relation to ascertaining proteins.
The problem with such diets are they are not to be taken lightly.
Diabetes is a disease who's symptoms include ants consuming the urine of those affected because of the urines sugar content.
Can you provide evidence of where an auto-immune disorder actually attacks, only a specific organ?
Multiple sclerosis (MS), also known as disseminated sclerosis or encephalomyelitis disseminata, is a demyelinating disease in which the insulating covers of nerve cells in the brain and spinal cord are damaged. This damage disrupts the ability of parts of the nervous system to communicate, resulting in a wide range of signs and symptoms,[1][2] including physical, mental,[2] and sometimes psychiatric problems.[3] MS takes several forms, with new symptoms either occurring in isolated attacks (relapsing forms) or building up over time (progressive forms).[4] Between attacks, symptoms may disappear completely; however, permanent neurological problems often occur, especially as the disease advances.[4]
While the cause is not clear, the underlying mechanism is thought to be either destruction by the immune system or failure of the myelin-producing cells.[5] Proposed causes for this include genetics and environmental factors such as infections.[2][6] MS is usually diagnosed based on the presenting signs and symptoms and the results of supporting medical tests.
Like many other diseases, multiple sclerosis existed long before it had a name. Writings from as far back as the Middle Ages described patients with symptoms that doctors today believe pointed to MS. A Dutch saint named Lidwina, who died in 1433, may have been one of the first known MS patients. After she fell while ice skating, she developed symptoms such as excruciating pain, blinding headaches, problems walking and paralysis. Her condition got worse over the course of her life, but she did have remissions. Another well-known potential MS patient was a grandson of King George III, who described his symptoms in a diary that he kept until his death in 1848.
Twenty years later, Dr. Jean-Martin Charcot became the first person credited with identifying multiple sclerosis as a disease. A female patient of his suffered an unusual combination of symptoms. He tried some of the typical treatments for other neurological disorders, such as electrical stimulation and injections of silver (which helped alleviate the symptoms of syphilis), but none of them worked. After his patient died, he dissected her brain and discovered the brain lesions. He called the disease sclerose en plaques. Myelin was discovered shortly afterwards, although its exact significance was unknown.