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Many people are freaking out about ebola, but the fact is that there’s no reason to panic because it spreads too slowly. Way slower than other infectious diseases. The graphic above shows it clearly: While a measles patient can infect a maximum of 18 people on average, an ebola patient can only infect two.
That’s what basic R0 means — the “maximum number of people who can catch the disease from one sick person, on average, in an outbreak” when “everyone in the population is susceptible to the disease.” This is also known as reproduction number or R nought.
Statistically, this means that the virus can easily be stopped in a highly developed country like the United States, ...
Do you think we might be facing the beginnings of a pandemic?
There will certainly be Ebola patients from Africa who come to us in the hopes of receiving treatment. And they might even infect a few people here who may then die. But an outbreak in Europe or North America would quickly be brought under control. I am more worried about the many people from India who work in trade or industry in west Africa. It would only take one of them to become infected, travel to India to visit relatives during the virus's incubation period, and then, once he becomes sick, go to a public hospital there. Doctors and nurses in India, too, often don't wear protective gloves. They would immediately become infected and spread the virus.
originally posted by: Iamthatbish
a reply to: TinkerHaus
I didn't consider this doom porn. I considered this a wake up call to my family about germs outside the home.
Have you read me type how surprised I was that because I read labels at the store I touch way more than I put in my cart?
I think most people are grossed out by others that have no sense of cleanliness in public, now I consider these people to be an actual threat to my families health.
I think most people are grossed out by others that have no sense of cleanliness in public, now I consider these people to be an actual threat to my families health.
originally posted by: ZiggyMojo
a reply to: cuckooold
I think the rate at which Ebola infects is dependent so far on geography and the population density. There is a very real chance of an airborne mutation that would skyrocket infection rates. Every time Ebola infects a new person, trillions of the virus are replicated, every one of those replications provides a chance for mutation. They've described over 50 mutations that we have witnessed since the outbreak began. As Ebola is exposed to the more densely populated areas, expect the infections per person number to go up. These things start slow, in the past 3 weeks there have been more infections than the entire outbreak leading up to those 3 weeks. It is an exponential equation. We are nearing the point where this can go one of two ways.. There is a window for us to operate in and be successful in stopping this spread. Some Doctors speculate that we may have already surpassed that window but many others feel we have time to slow this down before we suffer more drastic casualties but the window is indeed closing. If this is not contained, we will face a scenario where we have to let the virus burn itself out. In other words, isolate it enough that it can run its course until all infected have died or beat the virus.
When it comes to viruses, it is always difficult to predict what they can or cannot do. It is instructive, however, to see what viruses have done in the past, and use that information to guide our thinking. Therefore we can ask: has any human virus ever changed its mode of transmission?
The answer is no. We have been studying viruses for over 100 years, and we’ve never seen a human virus change the way it is transmitted.
HIV-1 has infected millions of humans since the early 1900s. It is still transmitted among humans by introduction of the virus into the body by sex, contaminated needles, or during childbirth.
Hepatitis C virus has infected millions of humans since its discovery in the 1980s. It is still transmitted among humans by introduction of the virus into the body by contaminated needles, blood, and during birth.
There is no reason to believe that Ebola virus is any different from any of the viruses that infect humans and have not changed the way that they are spread.
We Can’t Stop the Ebola Epidemic Unless We Understand How It’s Spread
Michael T. Osterholm – director of the Center for Infectious Disease Research and Policy at the University of Minnesota – wrote in the New York Times last month:
Viruses like Ebola are notoriously sloppy in replicating, meaning the virus entering one person may be genetically different from the virus entering the next. The current Ebola virus’s hyper-evolution is unprecedented; there has been more human-to-human transmission in the past four months than most likely occurred in the last 500 to 1,000 years. Each new infection represents trillions of throws of the genetic dice.
originally posted by: TinkerHaus
a reply to: boncho
boncho,
I love reading your posts and I respect your opinion, but I disagree with you that we should all be freaking out.
People keep saying "if it goes airborne..." but we have never witnessed a virus going airborne. Most of these viruses have existed in animal populations for millions of years, and have not gone airborne. Can you give any examples of viruses that humanity has ever witnessed "going airborne?"
The movie Outbreak ruined all of you. THIS IS NOT THE MOVIES.
originally posted by: TinkerHaus
a reply to: boncho
This is an example of engineering a virus to infect a different species - this is not an example of a virus naturally mutating to "go airborne."
Airborne transmission of the flu virus can be dependent on relative humidity and temperature, with colder, dry weather enabling the spread of the virus most easily.
Many common infections can spread by airborne transmission at least in some cases, including: Anthrax (inhalational), Chickenpox, Influenza, Measles, Smallpox and Tuberculosis.
Coughing, sneezing, talking, bed-making, turning pages of books, etc. all generate microbial aerosols which are carried and dispersed by air movements. Inhalation of these particles may cause allergic responses but whether or not infectious disease ensues depends in part on the viability and infectivity of the inhaled microbes and their landing sites. Desiccation is experienced by all airborne microbes; gram-negative bacteria and lipid-containing viruses demonstrate phase changes in their outer phospholipid bilayer membranes owing to concomitant changes in water content and/or temperature. These changes most likely lead to cross-linking reactions of associated protein moieties principally at mid to high relative humidity (RH). For lipid-free viruses these reactions of their surface protein moieties occur most rapidly at low RH. Radiation, oxygen, ozone and its reaction products and various pollutants also decrease viability and infectivity through chemical, physical and biological modification to phospholipid, protein and nucleic acid moieties. The extent of damage and the degree of repair together with the efficacy of host defence mechanisms largely controls whether the causative microbes take hold and spread disease via the airborne route. At least indoors, where desiccation is the predominant stress, the general reversibility of membrane-phase changes by vapour-phase rehydration when coupled with efficacious microbial enzymatic repair mechanisms under genetic control, virtually ensures the spread of disease by the aerobiological pathway.