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originally posted by: GetHyped
This is nonsense. You don't get the flu from the flu vaccine. The flu vaccine is not 100% effective which is why even if youg et the jab you can still get the flu.
originally posted by: ElectricUniverse
a reply to: HawkeyeNation
I am sure you missed this part.
In addition to the violation of personal rights, the CDC published a study in 2013 showing that vaccination of healthcare workers with the seasonal flu vaccine offered no significant measurable protection of patients from the flu. (See: CDC Study: Mandatory Flu Vaccinations of Health Care Workers Offer NO Protection to Patients)
Background
Vaccination of health-care workers has been claimed to prevent nosocomial influenza infection of elderly patients in long-term care. Data are, however, limited on this strategy. We aimed to find out whether vaccination of healthcare workers lowers mortality and the frequency of virologically proven influenza in such patients.
Methods
In a parallel-group study, health-care workers in 20 long-term elderly-care hospitals (range 44—105 patients) were randomly offered or not offered influenza vaccine (cluster randomisation, stratified for policy for vaccination of patients and hospital size). All deaths among patients were recorded over 6 months in the winter of 1996—97. We selected a random sample of 50% of patients for virological surveillance for influenza, with combined nasal and throat swabs taken every 2 weeks during the epidemic period. Swabs were tested by tissue culture and PCR for influenza viruses A and B.
Findings
Influenza vaccine uptake in health-care workers was 50·9% in hospitals in which they were routinely offered vaccine, compared with 4·9% in those in which they were not. The uncorrected rate of mortality in patients was 102 (13·6%) of 749 in vaccine hospitals compared with 154 (22·4%) of 688 in no-vaccine hospitals (odds ratio 0·58 [95% CI 0·40—0·84], p=0·014). The two groups did not differ for proportions of patients positive for influenza infection (5·4% and 6·7%, respectively); at necropsy, PCR was positive in none of 17 patients from vaccine hospitals and six (20%) of 30 from novaccine hospitals (p=0·055).
Interpretation
Vaccination of health-care workers was associated with a substantial decrease in mortality among patients. However, virological surveillance showed no associated decrease in non-fatal influenza infection in patients.
The vaccine is made from an inactivated virus that can't transmit infection. So people who get sick after receiving a flu vaccination were going to get sick anyway. It takes a week or two to get protection from the vaccine. But people assume that because they got sick after getting the vaccine, the shot caused their illness.
New Canadian studies suggest seasonal flu shot increased H1N1 risk
Filed Under: H1N1 2009 Pandemic Influenza; Influenza Vaccines
By: Maryn McKenna | Apr 06, 2010
Apr 6, 2010 (CIDRAP News) – Despite a rapidly launched range of studies, investigators in Canada are still unable to say—or to rule out—whether receiving a seasonal flu vaccination in the 2008-09 season made it more likely that Canadians would become ill from 2009 pandemic H1N1 flu.
In a lengthy article published today in Public Library of Science Medicine (PLoSMed), researchers detail the results of four supplementary studies that were launched after an April 2009 school outbreak provided the first signal of an association between seasonal flu shots and pandemic flu illness. The studies, which took in about 2,700 people, found overall that the likelihood of needing medical attention for pandemic flu was 1.4 to 2.5 times greater among people who were vaccinated the previous fall.
But the authors warn that, since all four studies were observational, even careful design cannot rule out the possibility that some undetected methodologic bias affected the results. That caution is echoed in a companion editorial, written by US researchers unconnected to the Canadian study, who cite the contradictory results of six other studies conducted in Mexico, Australia, and the United States at the same time as the Canadian ones. Four of those studies found no association between seasonal flu vaccination and pandemic flu illness, while the two done in Mexico paradoxically found that seasonal flu shots may have had a protective effect.
The Canadian researchers add, however, that their results may signal a heretofore-undetected biological mechanism of interaction, one that could occur again in the rare instance of a pandemic strain arising after a flu season has already begun. The 2009 pandemic strain surfaced in the last weeks of the 2008-09 flu season, months after vaccines for that season had been administered.
"Our results may seem counterintuitive, but they cannot be dismissed on the basis that no biological mechanism can plausibly explain them," the Canadian researchers write. "If these observations do reflect a real biological effect . . . they raise important questions that warrant further scientific observation."
...
Analysis finds limited evidence for HCW flu vaccination
Filed Under: Influenza Vaccines
Lisa Schnirring | Staff Writer | CIDRAP News | Sep 19, 2013
Hospitals and public health officials strongly promote healthcare worker (HCW) flu vaccination as a step to protect patients, but a new analysis found that evidence for a benefit isn't as strong as previously thought.
And influenza experts who commented on the analysis pointed out that, for specific outcomes such as lab-confirmed influenza, the data showed little evidence of protection for patients. They agreed, though, that immunization is a good measure to take.
The meta-analysis, by researchers from the US Centers for Disease Control and Prevention (CDC), appeared in an early online edition of Clinical Infectious Diseases. They focused on four randomized controlled trials and four observational studies from long-term facilities or hospitals. They pooled the results and assigned grades to the quality of the evidence.
...
originally posted by: ElectricUniverse
originally posted by: GetHyped
This is nonsense. You don't get the flu from the flu vaccine. The flu vaccine is not 100% effective which is why even if youg et the jab you can still get the flu.
IT's not nonsense. When you inject yourself with the vaccine you are injecting yourself the virus, or bacteria that is supposedly in a weakened state. Even if it is weakened you are still injecting yourself with the virus, and this compromises your immune system. Because of this, more so people with an already weakened immune system, will get sick.
originally posted by: GetHyped
This is nonsense. You don't get the flu from the flu vaccine. The flu vaccine is not 100% effective which is why even if youg et the jab you can still get the flu.
by Randall Neustaedter, O.M.D., L.Ac.
...
Could vaccines be weakening the immune system of our populations and causing recurrent infections and allergies at unprecedented levels? The only event that all infants routinely encounter at two months of age is vaccination with at least five different vaccines (Diphtheria-Tetanus-Pertussis-Polio-Haemophilus). They are repeated at four months. Could this simple fact explain the onset of the recurrent illnesses that plague so many infants? If vaccines stimulate antibody production to fight diseases, why would they weaken the immune system? Is there any evidence that vaccines do cause illness and immune system dysfunction?
One answer came in a careful study of illness patterns observed in babies before and after vaccination, published in Clinical Pediatrics in 1988. If vaccines cause a weakened immune system, then we would expect to see a higher incidence of illness following vaccination. In that study conducted in Israel, the incidence of acute illnesses in the 30-day period following DTP vaccine was compared to the incidence in the same children for the 30-day period prior to vaccine. The three-day period immediately following vaccine was excluded because children frequently develop fever as a direct response to vaccine toxins. A total of 82 healthy infants received DTP, and their symptoms were reported by parents and observed by a pediatrician at weekly intervals. Those babies experienced a dramatic increase in fever, diarrhea, and cough in the month following DTP vaccine compared to their health before the shot.
How do researchers investigate immune system reactions to vaccines? First, they can observe the incidence of serious disease onset soon after vaccination. They can also study immune functions following vaccines given to children and adults. Two research models have been used to discover the possible adverse effect of vaccines on the immune system. Laboratory researchers observe whether vaccines have any negative effect on white blood cells, the body’s primary immune defense system. Clinical researchers study illness patterns preceding and following vaccination. All of these investigative channels have reached the same conclusions—vaccines can trigger immune system suppression.
Vaccines are destroying our immune systems. Amazingly, the medical profession ignores the incriminating evidence against vaccines, and continues to inflict more unnecessary and harmful vaccines on our nation’s infants. A recent study from the New England Journal of Medicine of May 1996 revealed that tetanus vaccine disables the immune system in HIV patients. Tetanus vaccination produced a drop in T cells in 10 of 13 patients, a classic sign of immune deficiency. HIV viral replication increased dramatically in response to tetanus vaccine. Finally, white blood cells from 7 of 10 uninfected individuals became more susceptible to HIV infection following tetanus vaccination. Despite these findings, the authors made no comment about the immune depleting effect of the vaccine.
...
Different Types of Vaccines
...
Vaccines are made using several different processes. They may contain live viruses that have been attenuated (weakened or altered so as not to cause illness); inactivated or killed organisms or viruses; inactivated toxins (for bacterial diseases where toxins generated by the bacteria, and not the bacteria themselves, cause illness); or merely segments of the pathogen (this includes both subunit and conjugate vaccines).
...
Vaccines and Immune Suppression
by Harold E Buttram, MD
Introduction
Very few today would question that we are dealing with increasing patterns of sickness in today’s children as compared with earlier generations. Neurobehavioral problems are epidemic including autism, learning disabilities, and attention deficit hyperactivity disorder. In my experience, when elementary school teachers have been questioned about this matter, answers have been unanimous and emphatic, that they are now seeing a much greater incidence of these disorders with almost visible increases by the year. The same can be said for allergies and general patterns of sickness.
Until recently some have contended that the increase in these disorders has been due to better diagnosis, but this is no longer the case as reflected by current Center for Disease Control statistics showing increases in each of these categories. What then are the causes of this ominous health trend in our children? There are clues which relate in part to vaccines. Epidemiologic studies from England, (1) Sweden, (2) Africa, (3) and New Zealand (4) have consistently shown a much greater incidence of atopic disorders and patterns of sickness in fully vaccinated children as compared to those with limited or no vaccines. For this reason it may be of interest to review some of the pertinent medical literature on adverse vaccine effects in this area.
Vaccines and Immune Impairment – a Representative Review of the Literature
In a Letter-to-the Editor to the New England Journal of Medicine (1984), Eibl et al reported on a study of routine tetanus booster immunizations in 11 healthy adults in which T-lymphocyte subpopulations (white blood cells which help govern the immune system) were tested before and after immunizations. (5) Special concern rests in the fact that in 4 of the subjects the T-helper lymphocytes temporarily dropped to levels found in active AIDS patients. Comment: If this was the result of a single vaccine in healthy adults, it is sobering to think of the immune consequences of the multiple vaccines given repeatedly to infants with their immature and vulnerable immune systems during their first 6 months of life; and yet, as far as I am aware, this test has never been repeated.
...
J Toxicol Environ Health A. 2011;74(14):903-16. doi: 10.1080/15287394.2011.573736.
A positive association found between autism prevalence and childhood vaccination uptake across the U.S. population.
Delong G.
Author information
Abstract
The reason for the rapid rise of autism in the United States that began in the 1990s is a mystery. Although individuals probably have a genetic predisposition to develop autism, researchers suspect that one or more environmental triggers are also needed. One of those triggers might be the battery of vaccinations that young children receive. Using regression analysis and controlling for family income and ethnicity, the relationship between the proportion of children who received the recommended vaccines by age 2 years and the prevalence of autism (AUT) or speech or language impairment (SLI) in each U.S. state from 2001 and 2007 was determined. A positive and statistically significant relationship was found: The higher the proportion of children receiving recommended vaccinations, the higher was the prevalence of AUT or SLI. A 1% increase in vaccination was associated with an additional 680 children having AUT or SLI. Neither parental behavior nor access to care affected the results, since vaccination proportions were not significantly related (statistically) to any other disability or to the number of pediatricians in a U.S. state. The results suggest that although mercury has been removed from many vaccines, other culprits may link vaccines to autism. Further study into the relationship between vaccines and autism is warranted.
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Do aluminum vaccine adjuvants contribute to the rising prevalence of autism?
Lucija Tomljenovic a,⁎, Christopher A. Shaw a,b
a Neural Dynamics Research Group, Department of Ophthalmology and Visual Sciences, University of British Columbia, 828 W. 10th Ave, Vancouver, BC, Canada V5Z 1L8
b Departments of Ophthalmology and Visual Sciences and Experimental Medicine and the Graduate Program in Neuroscience, University of British Columbia, Vancouver, British Columbia,
828 W. 10th Ave, Vancouver, BC, Canada V5Z 1L8
a b s t r a c t
Autism spectrum disorders (ASD) are serious multisystem developmental disorders and an urgent global
public health concern. Dysfunctional immunity and impaired brain function are core deficits in ASD. Aluminum
(Al), the most commonly used vaccine adjuvant, is a demonstrated neurotoxin and a strong immune stimulator.
Hence, adjuvant Al has the potential to induce neuroimmune disorders. When assessing adjuvant toxicity in children,
two key points ought to be considered: (i) children should not be viewed as “small adults” as their unique
physiology makes them much more vulnerable to toxic insults; and (ii) if exposure to Al from only few vaccines
can lead to cognitive impairment and autoimmunity in adults, is it unreasonable to question whether the current
pediatric schedules, often containing 18 Al adjuvanted vaccines, are safe for children? By applying Hill's criteria
for establishing causality between exposure and outcome we investigated whether exposure to Al from vaccines
could be contributing to the rise in ASD prevalence in the Western world. Our results show that: (i) children from
countries with the highest ASD prevalence appear to have the highest exposure to Al from vaccines; (ii) the increase
in exposure to Al adjuvants significantly correlates with the increase in ASD prevalence in the United
States observed over the last two decades (Pearson r=0.92, pb0.0001); and (iii) a significant correlation exists
between the amounts of Al administered to preschool children and the current prevalence of ASD in seven Western
countries, particularly at 3–4 months of age (Pearson r=0.89–0.94, p=0.0018–0.0248). The application of
the Hill's criteria to these data indicates that the correlation between Al in vaccines and ASD may be causal. Because
children represent a fraction of the population most at risk for complications following exposure to Al, a
more rigorous evaluation of Al adjuvant safety seems warranted.
...
J Toxicol Environ Health A. 2010;73(24):1665-77. doi: 10.1080/15287394.2010.519317.
Hepatitis B vaccination of male neonates and autism diagnosis, NHIS 1997-2002.
Gallagher CM1, Goodman MS.
Author information
Abstract
Universal hepatitis B vaccination was recommended for U.S. newborns in 1991; however, safety findings are mixed. The association between hepatitis B vaccination of male neonates and parental report of autism diagnosis was determined. This cross-sectional study used weighted probability samples obtained from National Health Interview Survey 1997-2002 data sets. Vaccination status was determined from the vaccination record. Logistic regression was used to estimate the odds for autism diagnosis associated with neonatal hepatitis B vaccination among boys age 3-17 years, born before 1999, adjusted for race, maternal education, and two-parent household. Boys vaccinated as neonates had threefold greater odds for autism diagnosis compared to boys never vaccinated or vaccinated after the first month of life. Non-Hispanic white boys were 64% less likely to have autism diagnosis relative to nonwhite boys. Findings suggest that U.S. male neonates vaccinated with the hepatitis B vaccine prior to 1999 (from vaccination record) had a threefold higher risk for parental report of autism diagnosis compared to boys not vaccinated as neonates during that same time period. Nonwhite boys bore a greater risk.
PMID: 21058170 [PubMed - indexed for MEDLINE]
J Biomed Sci. 2002 Jul-Aug;9(4):359-64.
Abnormal measles-mumps-rubella antibodies and CNS autoimmunity in children with autism.
Singh VK1, Lin SX, Newell E, Nelson C.
Author information
Abstract
Autoimmunity to the central nervous system (CNS), especially to myelin basic protein (MBP), may play a causal role in autism, a neurodevelopmental disorder. Because many autistic children harbor elevated levels of measles antibodies, we conducted a serological study of measles-mumps-rubella (MMR) and MBP autoantibodies. Using serum samples of 125 autistic children and 92 control children, antibodies were assayed by ELISA or immunoblotting methods. ELISA analysis showed a significant increase in the level of MMR antibodies in autistic children. Immunoblotting analysis revealed the presence of an unusual MMR antibody in 75 of 125 (60%) autistic sera but not in control sera. This antibody specifically detected a protein of 73-75 kD of MMR. This protein band, as analyzed with monoclonal antibodies, was immunopositive for measles hemagglutinin (HA) protein but not for measles nucleoprotein and rubella or mumps viral proteins. Thus the MMR antibody in autistic sera detected measles HA protein, which is unique to the measles subunit of the vaccine. Furthermore, over 90% of MMR antibody-positive autistic sera were also positive for MBP autoantibodies, suggesting a strong association between MMR and CNS autoimmunity in autism. Stemming from this evidence, we suggest that an inappropriate antibody response to MMR, specifically the measles component thereof, might be related to pathogenesis of autism.
Copyright 2002 National Science Council, ROC and S. Karger AG, Basel
PMID: 12145534 [PubMed - indexed for MEDLINE]
© 2004 American Society for Clinical Nutrition
Metabolic biomarkers of increased oxidative stress and impaired methylation capacity in children with autism1,2
S Jill James,
Paul Cutler,
Stepan Melnyk,
Stefanie Jernigan,
Laurette Janak,
David W Gaylor, and
James A Neubrander
Abstract
Background: Autism is a complex neurodevelopmental disorder that usually presents in early childhood and that is thought to be influenced by genetic and environmental factors. Although abnormal metabolism of methionine and homocysteine has been associated with other neurologic diseases, these pathways have not been evaluated in persons with autism.
Objective: The purpose of this study was to evaluate plasma concentrations of metabolites in the methionine transmethylation and transsulfuration pathways in children diagnosed with autism.
Design: Plasma concentrations of methionine, S-adenosylmethionine (SAM), S-adenosylhomocysteine (SAH), adenosine, homocysteine, cystathionine, cysteine, and oxidized and reduced glutathione were measured in 20 children with autism and in 33 control children. On the basis of the abnormal metabolic profile, a targeted nutritional intervention trial with folinic acid, betaine, and methylcobalamin was initiated in a subset of the autistic children.
Results: Relative to the control children, the children with autism had significantly lower baseline plasma concentrations of methionine, SAM, homocysteine, cystathionine, cysteine, and total glutathione and significantly higher concentrations of SAH, adenosine, and oxidized glutathione. This metabolic profile is consistent with impaired capacity for methylation (significantly lower ratio of SAM to SAH) and increased oxidative stress (significantly lower redox ratio of reduced glutathione to oxidized glutathione) in children with autism. The intervention trial was effective in normalizing the metabolic imbalance in the autistic children.
Conclusions: An increased vulnerability to oxidative stress and a decreased capacity for methylation may contribute to the development and clinical manifestation of autism.
...
Impaired transsulfuration and oxidative stress in ASD: Improvement with targeted nutrition
S. Jill James, PhD
The generation of reactive oxygen species (ROS) is an inevitable consequence of aerobic energy metabolism. A delicate oxidant-antioxidant balance within each cell maintains normal physiologic levels of ROS that serve as important signaling molecules for the activation of cGMP-dependent functions, for vasomotor tone, T cell activation, and normal gene expression 1,2. Oxidative stress occurs when cellular antioxidant defense mechanisms fail to counterbalance and control ROS production. Unopposed ROS can result in damage to mitochondrial and nuclear DNA, alteration in protein structure, and membrane lipid composition 3. Functionally, these aberrations translate into abnormal gene expression, membrane signal transduction, and altered rates of cell proliferation, differentiation, and apoptosis. A pro-oxidant microenvironment has been implicated in the etiology of numerous human diseases including cardiovascular disease, cancer, autoimmune disorders, and neurodegenerative conditions 4-8. The potential role of oxidative stress in the etiology of autism has received less research attention.
Overview of the Methionine Transsulfuration Pathway
...
M. Catherine DeSoto, PhD Department of Psychology, University of Northern Iowa, Cedar Falls, Iowa, [email protected]
Robert T. Hitlan, PhD Department of Psychology, University of Northern Iowa, Cedar Falls, Iowa
Abstract
The question of what is leading to the apparent increase in autism is of great importance. Like the link between aspirin and heart attack, even a small effect can have major health implications. If there is any link between autism and mercury, it is absolutely crucial that the first reports of the question are not falsely stating that no link occurs. We have reanalyzed the data set originally reported by Ip et al. in 2004 and have found that the original p value was in error and that a significant relation does exist between the blood levels of mercury and diagnosis of an autism spectrum disorder. Moreover, the hair sample analysis results offer some support for the idea that persons with autism may be less efficient and more variable at eliminating mercury from the blood.
...
Vaccines and Autism
Bernard Rimland, PhD, Woody McGinnis, MD
Autism Research Institute, San Diego, CA
Vaccinations may be one of the triggers for autism. Substantial data demonstrate immune abnormality in many autistic children consistent with impaired resistance to infection, activation of inflammatory response, and autoimmunity.
Impaired resistance may predispose to vaccine injury in autism.
A mercurial preservative in childhood vaccines, thimerosal, may cause direct neurotoxic, immunodepressive, and autoimmune injury and contribute to early onset and regressed autism. Live viruses in measles, mumps, and rubella (MMR) may result in chronic infection of the gut and trigger regressed autism. Thimerosal injection may potentiate MMR injury.
Consideration of vaccine etiology must include recognition of compromised gut and nutrition in most autistic children.
An integrated view of the underlying biological problems in autistic children serves our understanding of the possible role of vaccines.
Development of screening methods for deferral of vaccines in atrisk
children is a worthy goal.
...
Neurotoxicology. 2005 Jan;26(1):1-8.
Thimerosal neurotoxicity is associated with glutathione depletion: protection with glutathione precursors.
James SJ1, Slikker W 3rd, Melnyk S, New E, Pogribna M, Jernigan S.
Author information
Abstract
Thimerosol is an antiseptic containing 49.5% ethyl mercury that has been used for years as a preservative in many infant vaccines and in flu vaccines. Environmental methyl mercury has been shown to be highly neurotoxic, especially to the developing brain. Because mercury has a high affinity for thiol (sulfhydryl (-SH)) groups, the thiol-containing antioxidant, glutathione (GSH), provides the major intracellular defense against mercury-induced neurotoxicity. Cultured neuroblastoma cells were found to have lower levels of GSH and increased sensitivity to thimerosol toxicity compared to glioblastoma cells that have higher basal levels of intracellular GSH. Thimerosal-induced cytotoxicity was associated with depletion of intracellular GSH in both cell lines. Pretreatment with 100 microM glutathione ethyl ester or N-acetylcysteine (NAC), but not methionine, resulted in a significant increase in intracellular GSH in both cell types. Further, pretreatment of the cells with glutathione ethyl ester or NAC prevented cytotoxicity with exposure to 15 microM Thimerosal. Although Thimerosal has been recently removed from most children's vaccines, it is still present in flu vaccines given to pregnant women, the elderly, and to children in developing countries. The potential protective effect of GSH or NAC against mercury toxicity warrants further research as possible adjunct therapy to individuals still receiving Thimerosal-containing vaccinations.
PMID: 15527868 [PubMed - indexed for MEDLINE]
J Immunotoxicol. 2013 Apr-Jun;10(2):210-22. doi: 10.3109/1547691X.2012.708366. Epub 2012 Sep 11.
How aluminum adjuvants could promote and enhance non-target IgE synthesis in a genetically-vulnerable sub-population.
Terhune TD1, Deth RC.
Author information
Abstract
Aluminum-containing adjuvants increase the effectiveness of vaccination, but their ability to augment immune responsiveness also carries the risk of eliciting non-target responses, especially in genetically susceptible individuals. This study reviews the relevant actions of aluminum adjuvants and sources of genetic risk that can combine to adversely affect a vulnerable sub-population. Aluminum adjuvants promote oxidative stress and increase inflammasome activity, leading to the release of IL-1β, IL-18, and IL-33, but not the important regulatory cytokine IL-12. In addition, they stimulate macrophages to produce PGE₂, which also has a role in regulating immune responses. This aluminum-induced cytokine context leads to a T(H)2 immune response, characterized by the further release of IL-3, IL-4, IL-5, IL-9, IL-13, and IgE-potentiating factors such as sCD23. Genetic variants in cytokine genes, such as IL-4, IL-13, IL-33, and IL-18 influence the response to vaccines in children and are also associated with atopy. These genetic factors may therefore define a genetically-vulnerable sub-population, children with a family history of atopy, who may experience an exaggerated T(H)2 immune response to aluminum-containing vaccines. IL-4, sCD23, and IgE are common factors for both atopy and the immune-stimulating properties of aluminum adjuvants. IL-4 is critical in the production of IgE and total IgE up-regulation. IL-4 has also been reported to induce the production of sCD23 and trigger resting sIgM+, sIgD+ B-cells to switch to sIgE+ B-cells, making them targets for IgE-potentiating factors. Further, the actions of IgE-potentiating factors on sIgE+ B-cells are polyclonal and unrestricted, triggering their differentiation into IgE-forming plasma cells. These actions provide a mechanism for aluminum-adjuvant promotion and enhancement of non-target IgE in a genetically vulnerable sub-population. Identification of these individuals may decrease the risk of adverse events associated with the use of aluminum-containing vaccines.
a reply to: tom.farnhill
i had an accident 60 years ago and i had to have my spleen removed , i was told that my immune system would be very low from then on .
a friend of mine also had the same operation when he was in his 40s and he has to take penicilin for the rest of his life .
i have never taken anything other than the tetanus shot for a bad cut that required stitches
i am now 66 years old and in very good health .