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Originally posted by TiredofControlFreaks
reply to post by OccamsRazor04
Did you ever actually read the Enstrom Study Here it is"
www.bmj.com...
Did your own quote not say that the ACS followed the the group for several decades?
Richar Smith, the editor and publisher of the BMJ that decided to publish the Enstrom study (and defended his decision to do so) RESIGNED his university post to protest the university accepting tobacco money
www.ash.org.uk...
This link has many newpaper articles announcing his decision.
Tired of Control Freaks
Originally posted by TiredofControlFreaks
reply to post by OccamsRazor04
Your point about non-smokers being MORE affected by smoke is wrong because tobacco is not the only source of smoke or nicotene.
Can you really think of anytime in a millenia of history when man has used fire to burn organic material to cook food and heat their homes? Is there even one human being on this planet who has never been exposed to smoke from a campfire, a candle, car exhaust, a fireplace, a wood burning stove and grilled food?
Baby boomers today were exposed a children when the incidence of smoking in the population was the highest.
Another way to interpret your point is that smokers are habituated to the contaminants in smoke and are therefore protected from the contaminants of other sources of smoke. Never-smokers have no such protection.
Wood Smoke vs. Cigarette Smoke Although many people associate tobacco smoke with certain health risks, research indicates that second hand wood smoke has potentially even greater ability to damage health. A comparison between tobacco smoke and wood smoke using electron spin resonance revealed quite startling results (Rozenberg 2001, Wood Smoke is More Damaging than Tobacco Smoke). Tobacco smoke causes damage in the body for approximately 30 seconds after it is inhaled. Wood smoke, however, continues to be chemically active and cause damage to cells in the body for up to 20 minutes, or 40 times longer.
Originally posted by TiredofControlFreaks
reply to post by TiredofControlFreaks
oh and BTW - the anti-smokers rely on another type of study for evidence that second hand smoke from tobacco is causing harm to the population. They do a retrospective study where they ask a person who has a disease if they were ever exposed to second hand smoke and rely on the person to be honest and to remember their lives accurately.
A little more open to bias and suggestion there, I would think.
Tired of Control freaks
Originally posted by TiredofControlFreaks
reply to post by OccamsRazor04
In response to your question about how 31 % increase in risk could not be significant.
Are you imagining something like - if there are 100 people and nobody smokes than only 1 person gets cancer but if they all smoke 31 people get cancer?
That is generally how people interpret an increase in relative risk. However that is NOT what it means.
Let me explain:
There is a very big difference in the meaning of the word "significant" among mathematicians and the ordinary use of the word (meaning a large change). Mathematics defines the word "significant" using math to calculate it. But just because something is mathematically significant does not mean that it is clinically significant.
Clinically significant means that the change in magnitude would definitively chance the occurence in the incidence of the disease in the population.
In epidimiology, any increase of risk that is less than 300 % and preferably 400 % is not considered to be clinically signicant.
en.wikipedia.org...
In the Enstrom study - a difference in relative risk of 31 % is mathematically significant but clinically insignificant (ie there would be no discernible increase in the rate of the disease among the general population)
Generally study start off with a hypothesis I.e. Does being exposed to second hand smoke because you are married to a smoke and you are a never smoker cause you to get lung cancer?
If the relative risk is over 300 % increased - the answer is maybe. (you still have to check for other causes of lung cancer like if the study area has a lot of radon or a lot of exposure to diesel fumes or if people cook on BBQ's alot)
If the relative risk is less than that - the answer is a no.
Of course you have to also get other scientists to do the same study to see if they come out with the same results.
Tired of Control Freaks
Belgium has established an ad hoc-committee to develop a comprehensive social marketing and professional training strategy for pilot testing preconception care models in the French speaking part of Belgium, an area that represents 5 million people and 50,000 births per year using prenatal care and pediatric clinics, gynecological departments, and the genetic centers.
Originally posted by TiredofControlFreaks
reply to post by OccamsRazor04
0h come on now - I am not going to do all your research for you!
10 % increase in relative risk is clinically significant - please provide your evidence for such a ridiculous statement?
Tired of Control Freaks
The standard level of significance used to justify a claim of a statistically significant effect is 0.05. For better or worse, the term statistically significant has become synonymous with P0.05.
For example, we may find that there is a statistically significant relationship between a citizen's age and satisfaction with city recreation services. It may be that older citizens are 5% less satisfied than younger citizens with city recreation services. But is 5% a large enough difference to be concerned about?
P
The pooled relative risk of heart disease in never-smokers exposed to secondhand smoke was 1.31, similar to estimates of earlier meta-analyses.
Results For participants followed from 1960 until 1998 the age adjusted relative risk (95% confidence interval) for never smokers married to ever smokers compared with never smokers married to never smokers was 0.94 (0.85 to 1.05) for coronary heart disease, 0.75 (0.42 to 1.35) for lung cancer, and 1.27 (0.78 to 2.08) for chronic obstructive pulmonary disease among 9619 men, and 1.01 (0.94 to 1.08), 0.99 (0.72 to 1.37), and 1.13 (0.80 to 1.58), respectively, among 25 942 women. No significant associations were found for current or former exposure to environmental tobacco smoke before or after adjusting for seven confounders and before or after excluding participants with pre-existing disease. No significant associations were found during the shorter follow up periods of 1960-5, 1966-72, 1973-85, and 1973-98.
If you are healthy, you're usually not at a major risk from short-term exposures to smoke.