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(Source, Source Source)
"In December 2006, a 12 year gateway drug hypothesis study on 214 boys from ages 10–12 by the American Psychiatric Association was published in the American Journal of Psychiatry. The study concluded adolescents who used cannabis prior to using other drugs, including alcohol and tobacco, were no more likely to develop a substance abuse disorder than subjects in the study who did not use cannabis prior to using other drugs."
(Source Source)
"In 2004, a study comparing cannabis users in San Francisco to those in Amsterdam was done to test the effects of the differing drug policies in the two cities on drug use patterns. The Netherlands has a drug policy of decriminalization in which cannabis can be bought by adults over 18 in quasi-legal "coffee shops" and used publicly, while in the United States cannabis is criminalized and must be bought in the black market (often from the same dealers that sell hard drugs) and used "underground". The results found that, compared with their counterparts in Amsterdam the San Francisco cannabis users were significantly more likely to use coc aine, crack, amphetamines, ecstasy, and opiates despite similar cannabis use patterns and a more permissive drug policy in the Netherlands. One plausible explanation is that the black market itself acts as a gateway to harder drugs, as opposed to the effects of cannabis per se."
Source
While marijuana went from being a secret shared by a small community of hepcats and beatniks in the 1940s and '50s to a rite of passage for some 70% of youth by the turn of the century, rates of schizophrenia in the U.S. have remained flat, or possibly declined. For as long as it has been tracked, schizophrenia has been found to affect about 1% of the population.
One explanation may be that the two factors are coincidental, not causal: perhaps people who have a genetic susceptibility to schizophrenia also happen to especially enjoy marijuana. Still, some studies suggest that smoking pot can actually trigger the disease earlier in individuals who are predisposed, and yet researchers still aren't seeing increases in the overall schizophrenia rate or decreases in the average age of onset.
Source
There have been many studies promoting the link between cannabis use and the onset of schizophrenia. It is not the quantity of scientific studies that matters though, it is the quality. As stated in some of my other marijuana myth posts there are three criteria that need to be satisfied in order to prove causality, these are: Association, Temporal Antecedence and Isolation. Association means the cause and effect must occur together, temporal antecedence means the effect must follow the cause and isolation means all other causes must be ruled out. The first two can be shown for pretty much anything.
For example if I performed a raindance and it rained later in the day I have satisfied association and temporal antecedence but not isolation. Therefore I cannot scientifically prove that my raindance worked. If raindances were a controversial topic the media would then publish the “study” and the headline would read “Recent Study Suggests that Raindance Leads to precipitation”. Sounds ridiculous right? Well that is exactly what has been happening with the hundreds of “studies” on cannabis and schizophrenia.
Isolation has not been proven in the marijuana-schizophrenia link. In fact evidence to the contrary is indisputable. Marijuana use has grown significantly in the last 30 years. If schizophrenia was caused by marijuana use then there should be an observable rise in schizophrenia rates as well. No such link has been established. This study from Australia demonstrates these findings (Source). Before marijuana became popular less than 1% of the population was diagnosed with schizophrenia, and now that marijuana use is widespread the rate of schizophrenia diagnosis is still less than 1%.
Source
[T]he expected rise in diagnoses of schizophrenia and psychoses did not occur over a 10 year period. This study does not therefore support the specific causal link between cannabis use and incidence of psychotic disorders. … This concurs with other reports indicating that increases in population cannabis use have not been followed by increases in psychotic incidence.
Source
No mental pathology directly related to the overuse of cannabis has been reported, which distinguishes this substance from psychostimulants such as MDNA, coc aine or alcohol, heavy and repeated use of which can give rise to characteristic psychotic syndromes. Similarly, cannabis does not seem to precipitate the onset of pre-existing mental dysfunctions (schizophrenia, bipolar depression, etc.).
Myth: "Parents are often unaware that today's marijuana is different from that of a generation ago, with potency levels 10 to 20 times stronger than the marijuana with which they were familiar." (US Drug Czar John Walters)
Myth: "Is marijuana a gateway drug?
One plausible explanation is that the black market itself acts as a gateway to harder drugs, as opposed to the effects of cannabis per se.