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Originally posted by MatrixProphet
reply to post by ExistenceUnknown
You said that naturalistic pertains to that which is non-supernatural.
This of course would depend completely on perception. Imagine that there is a Super Scientific God in the sky (which I believe in) who in his perception is natural and even may come from a family that would consider themselves "natural."
Originally posted by yeti101
reply to post by platosallegory
so how do ID folks propose intelligent design works?
Yes in the context I used it that is what I meant. I do not contend that nature is the only thing that exists. Rather, nature is the only objective standard we have because the supernatural has never been reliably observed.
Originally posted by thesdoc
Originally posted by stander
Abiogenesis comprise two parts: ABIO and GENESIS. "Abio" is made of A,B, I and O that somewhat fused together and formed the word "abio," which doesn't make sense -- unless I have a very thin dictionary.
Hello, I am not judging or taking sides here, I just wanted to point out that the abio part has a very well defined and non-ambiguous meaning.
A - BIO means without life. A is "a" negative in ancient greek. Bios (Βίος) = life (ζωή).
Abiogenesis (Αβιογένεσις) = Birth without life. So the term is used to define the moment life emerged out of lifeless organic matter.
O Stokos.
Originally posted by ExistenceUnknown
Evolution is fact, if anyone out there could prove that it was indeed not fact then you deserve fame and fortune. If evidence was found to prove that evolution is false under lab scrutiny then it would be welcomed with open arms into the scientific community.
"Don't get me wrong, im not saying its 100% fact.- ExistenceUnknown"
"In other words Scientific Theory is the Politically correct way of saying "Fact". That's the beauty of science. It will not claim anything a fact if there is a .000000001 chance that something could prove it wrong.- ExistenceUnknown
www.abovetopsecret.com...
"Why do people think there are no transitional fossils?
Ambulocetus
Archaeopteryx
Tiktaalik - existence unknown. "
Tiktaalik: Our Ancestor?
Apr 11, 2006
by Frank Sherwin
With the continued invalidation of the corrupt theory of neo-Darwinism in the eyes of many, and school boards nation-wide taking a favorable look at intelligent design, it is not surprising that evolutionists are scrambling to enact damage control. Enter an alleged “missing link” that some are saying reveals one of the greatest changes in the field of zoology.
The New York Times (NYT) reports that the recent discovery of a large scaly creature in Canada is “a predecessor of amphibians, reptiles and dinosaurs, mammals and eventually humans” (Wilford 2006). National Geographic News (NGN) crows that “fossil hunters may have discovered the fish that made humans possible.” (Owen 2006). But before evolutionists start celebrating, they should keep in mind that Tiktaalik roseae is incomplete. Scientists as of yet unable to determine what the hind fins and tail might have looked like. Paleontologist Neil Shubin states, “We’ve really only begun to sort of crack that spot [the small rocky outcropping 600 miles from the North Pole where Tiktaalik was found]” (AP 2006).
Also noteworthy, is the use of diffident language by the secular reporters and scientists when discussing Tiktaalik. For example, NGN says this creature “may” be a missing link. While the NYT states that changes in this creature “anticipate” the emergence of land animals. One may anticipate leaving the house, but he is still in the house.
There are five transposable elements on the pOAD2 plasmid. When activated, transposase enzymes coded therein cause genetic recombination. Externally imposed stress such as high temperature, exposure to a poison, or starvation can activate transposases. The presence of the transposases in such numbers on the plasmid suggests that the plasmid is designed to adapt when the bacterium is under stress.
2. All five transposable elements are identical, with 764 base pairs (bp) each. This comprises over eight percent of the plasmid. How could random mutations produce three new catalytic/degradative genes (coding for EI, EII and EIII) without at least some changes being made to the transposable elements? Negoro speculated that the transposable elements must have been a ‘late addition’ to the plasmids to not have changed. But there is no evidence for this, other than the circular reasoning that supposedly random mutations generated the three enzymes and so they would have changed the transposase genes if they had been in the plasmid all along. Furthermore, the adaptation to nylon digestion does not take very long (see point 5 below), so the addition of the transposable elements afterwards cannot be seriously entertained.
3. All three types of nylon degrading genes appear on plasmids and only on plasmids. None appear on the main bacterial chromosomes of either Flavobacterium or Pseudomonas. This does not look like some random origin of these genes—the chance of this happening is exceptionally low. If the genome of Flavobacterium is about two million bp, and the pOAD2 plasmid comprises 45,519 bp, and if there were say 5 pOAD2 plasmids per cell (~10% of the total chromosomal DNA), then the chance of getting all three of the genes on the pOAD2 plasmid would be about 0.0015. If we add the probability of the nylon degrading genes of Pseudomonas also only being on plasmids, the probability falls to 2.3 x 10-6. If the enzymes developed in the independent laboratory-controlled adaptation experiments (see point 5, below) also resulted in enzyme activity on plasmids (almost certainly, but not yet determined), then attributing the development of the adaptive enzymes purely to chance mutations becomes even more implausible.
4. The antisense DNA strand of the four nylon genes investigated in Flavobacterium and Pseudomonas lacks any stop codons. This is most remarkable in a total of 1,535 bases. The probability of this happening by chance in all four antisense sequences is about 1 in 1012. Furthermore, the EIII gene in Pseudomonas is clearly not phylogenetically related to the EII genes of Flavobacterium, so the lack of stop codons in the antisense strands of all genes cannot be due to any commonality in the genes themselves (or in their ancestry). Also, the wild-type pOAD2 plasmid is not necessary for the normal growth of Flavobacterium, so functionality in the wild-type parent DNA sequences would appear not to be a factor in keeping the reading frames open in the genes themselves, let alone the antisense strands.
Some statements by Yomo et al., [Yomo T, Urabe I, Okada H (May 1992) “No stop codons in the antisense strands of the genes for nylon oligomer degradation”. Proc Natl Acad Sci USA. 89 (9): 3780–4.] express their utter consternation at the findings:
“These results imply that there may be some unknown mechanism behind the evolution of these genes for nylon oligomer-degrading enzymes.
“The presence of a long NSF (non-stop frame) in the antisense strand seems to be a rare case, but it may be due to the unusual characteristics of the genes or plasmids for nylon oligomer degradation.
“Accordingly, the actual existence of these NSFs leads us to speculate that some special mechanism exists in the regions of these genes.”
It looks like recombination of codons (base pair triplets), not single base pairs, has occurred between the start and stop codons for each sequence. This would be about the simplest way that the antisense strand could be protected from stop codon generation. The mechanism for such a recombination is unknown, but it is highly likely that the transposase genes are involved.
Interestingly, Yomo et al. also show that it is highly unlikely that any of these genes arose through a frame shift mutation, because such mutations (forward or reverse) would have generated lots of stop codons. This nullifies the claim of Thwaites that a functional gene arose from a purely random process (an accident).
5. The Japanese researchers demonstrated that nylon degrading ability can be obtained de novo in laboratory cultures of Pseudomonas aeruginosa [strain] POA, which initially had no enzymes capable of degrading nylon oligomers. This was achieved in a mere nine days! The rapidity of this adaptation suggests a special mechanism for such adaptation, not something as haphazard as random mutations and selection.
6. The researchers have not been able to ascertain any putative ancestral gene to the nylon-degrading genes. They represent a new gene family. This seems to rule out gene duplications as a source of the raw material for the new genes.
P. aeruginosa is renowned for its ability to adapt to unusual food sources—such as toluene, naphthalene, camphor, salicylates and alkanes. These abilities reside on plasmids known as TOL, NAH, CAM, SAL and OCT respectively. Significantly, they do not reside on the chromosome (many examples of antibiotic resistance also reside on plasmids).The chromosome of P. aeruginosa has 6.3 million base pairs, which makes it one of the largest bacterial genomes sequenced. Being a large genome means that only a relatively low mutation rate can be tolerated within the actual chromosome, otherwise error catastrophe would result. There is no way that normal mutations in the chromosome could generate a new enzyme in nine days and hypermutation of the chromosome itself would result in non-viable bacteria. Plasmids seem to be adaptive elements designed to make bacteria capable of adaptation to new situations while maintaining the integrity of the main chromosome.
Now, to look at stasis in bacteria: P. aeruginosa was first named by Schroeter in 1872. It still has the same features that identify it as such. So, in spite of being so ubiquitous, so prolific and so rapidly adaptable, this bacterium has not evolved into a different type of bacterium. Note that the number of bacterial generations possible in over 130 years is huge—equivalent to tens of millions of years of human generations, encompassing the origin of the putative common ancestor of ape and man, according to the evolutionary story, indeed perhaps even all primates. And yet the bacterium shows no evidence of directional change—
stasis rules, not progressive evolution.
www.uncommondescent.com...
Originally posted by MatrixProphet
You would like me to give a 2+2 = 4 dissertation on this, using facts so you can stump me, but it doesn't work that way.
Originally posted by MatrixProphet
Either you have access to higher consciousness or you do not.
Originally posted by MatrixProphet
It would be like trying to explain the laws of physics to a 3 year old.
Originally posted by yeti101
If i went back in time what would i see at the point the designer designed a new species? I mean would there be nothing then a puff of smoke and a new species would appear? is that what i would see?
Originally posted by yeti101
thanks to those who tried to explain how ID works. But the answers i got wasnt what i was lookinG for. I'll try put it another way.
If i went back in time what would i see at the point the designer designed a new species? I mean would there be nothing then a puff of smoke and a new species would appear? is that what i would see?
thats what i mean by "how does it work?"
[edit on 22-5-2009 by yeti101]
Originally posted by ExistenceUnknown
Since I am not responding to this "Con Science" person due to his ridiculous debate tactics, I will leave it to say to the rest of you that anyone who digs through past posts to find things to justify their reasoning without at least linking to the original thread is a buffoon.
"In other words Scientific Theory is the Politically correct way of saying "Fact". That's the beauty of science. It will not claim anything a fact if there is a .000000001 chance that something could prove it wrong.- ExistenceUnknown
www.abovetopsecret.com...
I will not defend myself to this guy,
The fact that he is still responding to my posts after I told him I would not debate him anymore says wonders.
Otherwise I hope most of you recognize this dishonest approach that he is taking.