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So I see another incapable of thinking for one’s self. Peddling the same lies that the eugenics tell. Population is not growing at the same rate it was 30 years ago.
originally posted by: Thrumbo
a reply to: ColeYounger
6.5 million people died from covid-19 globally, just under 1% of the 8 billion of us, the total population of the planet. A "real" pandemic would be more like 10% of the species or more.
Of those 6.5 million, the vast majority were elderly or had poor health. Nature did us a favor? That's such a weak culling of the herd that you can't even call it that.
Did you know that the global population has more than doubled since 1960? There was only 3 billion humans back then, a mere 60 years ago. If this trend continues, in 2080 there will be over 16+ billion human beings. More than that, the equation is exponential until there aren't enough resources to support life. If we have supply chain issues now, imagine what they'll be like in the future.
In the year 1700, there was approximately 610 million people on the planet. Our population has increased over 10x.
originally posted by: chr0naut
originally posted by: asabuvsobelow
a reply to: chr0naut
If the SARS-CoV-2 virus had happened in 1918, there would have been about the same number of fatalities that the 1918 flu caused.
You must be joking ? Do you really think the people in 1918 would have even noticed a chest cold ? Covid-19 statistically killed almost no one chr0 and you know it , Those death numbers are wildly inaccurate .
The 1918 Flu on the other hand killed millions out right burning them up with fevers, it didn't matter if they were old , fat or had bad hearts .
You are hanging on your Covid-19 Talking points like they are actually true mate , The Curtain is lifting on the Plandemic mate it was a joke I suggest you pull chute while you can
Last week in New Zealand, COVID-19 caused the death of 37 people, one of whom was 10 years old, and two of whom were in the 15-35 year age bracket, out of an average of 9,611 active cases.
When people die of stuff, it is easily detectable. Even back in 1918, it was the case.
originally posted by: Asmodeus3
a reply to: chr0naut
I haven't followed the conversation but a quick comment.
SARS-CoV-2 has an infection fatality rate of about 0.15%
pubmed.ncbi.nlm.nih.gov...
On the other hand the Spanish Flu had an infection fatality rate of at least 10%
www.cdc.gov...
Two very different diseases COVID-19 and Spanish Flu.
At that time and according to the CDC around 500 million people were infected and at least 50 million died. If this has happened with a population of 8 billion and let's say one third of the population was infected then you were looking at least for 265 million deaths.
Best way to compare them is the IFR
The Spanish Flu has an IFR which is at least 67 times more than the IFR of COVID-19 and could be 100 times more lethal.
Overall, our meta - analysis fails to confirm the notion that lockdowns – at least in the spring of 2020 – had a large , significant effect on mortality rates.
Studies examining the relationship between lockdown strictness (based on the OxCGRT stringency index) find that the average lockdown in Europe and the United States only reduced COVID - 19 mortality by 3.2 % compared to the most lenient COVID - 19 policy .
Shelter - in- place orders (SIPO s ) were also ineffective . They only reduced COVID - 19 mortality by 2 . 0 % . Based on nine specific NPIs , we estimate that the average lockdown in Europe and the United States in the spring of 2020 reduced mortality by 10.7 %. The 3.2% to 10.7 % correspond 6,000 - 23 , 000 avoided deaths in Europe and 4,000 - 16 ,000 avoided deaths in the United States .
In comparison, there are approximately 72,000 flu deaths in Europe and 38,000 flu deaths in the United States each year. 77 Thus, lockdowns in Europe and the United States on average saved lives correspond ing to 9% - 35% of an average flu season .
Of the spec ific NPIs, w e find that mask mandates had the largest effect ( reducing COVID - 19 mortality by 1 8 . 7 %) , but the estimate is based on just three studies with heterogeneity in the definition of the mandate . Limiting gatherings were counterproductive and increased mortality by 5.9%.
Our measured meta - results are supported by the natural experiments we have been able to identify through our work and by searches i n the abstract and citation database Scopus (see Table 17 ) .
Overall, our meta - analysis support s the conclusion that lockdowns – at least in the spring of 2020 – had little to no effect on COVID - 19 mortality
originally posted by: asabuvsobelow
wow 37 people lets lock down the world again.
originally posted by: chr0naut
originally posted by: asabuvsobelow
a reply to: chr0naut
You must be joking ? Do you really think the people in 1918 would have even noticed a chest cold ? Covid-19 statistically killed almost no one chr0 and you know it , Those death numbers are wildly inaccurate .
If the SARS-CoV-2 virus had happened in 1918, there would have been about the same number of fatalities that the 1918 flu caused.
The 1918 Flu on the other hand killed millions out right burning them up with fevers, it didn't matter if they were old , fat or had bad hearts .
You are hanging on your Covid-19 Talking points like they are actually true mate , The Curtain is lifting on the Plandemic mate it was a joke I suggest you pull chute while you can
Last week in New Zealand, COVID-19 caused the death of 37 people, one of whom was 10 years old, and two of whom were in the 15-35 year age bracket, out of an average of 9,611 active cases.
When people die of stuff, it is easily detectable. Even back in 1918, it was the case.
2,200 people die every day of heart attacks.
2,380 people die every day from Cardiovascular disease .
1,670 people die every day from Cancer.
originally posted by: Asmodeus3
a reply to: chr0naut
The IFR is the most important number in trying to understand a disease and take decisions about public policies. It is estimated using surveillance and seroprevalence data and although it is an estimate there is a very good degree of accuracy in predicting how many people have been infected in total. That's why there are several peer reviewed scientific publications on the subject. The one by John Ioannidis is one of the most cited papers in the world, if not the most cited one, and hence it is a recognition of the work done by him and his colleagues. It stands as 0.15% approximately and it shows that COVID-19 is a mild disease for most people. In a few words we are expecting let's say 15 deaths in every 10,000 infections.
As in my previous post the Spanish Flu had an IFR of at least 10% and you can easily make the comparison.
pubmed.ncbi.nlm.nih.gov...
In conclusion and going almost to the third year after SARS-CoV-2 was discovered we can see that COVID-19 indeed has a very small infection fatality rate. By now most of the populations have been infected.
originally posted by: chr0naut
The 1918 flu had the almost the same case fatality ratio (CFR) as COVID-19 did prior to the roll-out of the vaccines (on average, the 1918 flu had a CFR slightly greater than 2.5%).
If the SARS-CoV-2 virus had happened in 1918, there would have been about the same number of fatalities that the 1918 flu caused.
originally posted by: Asmodeus3
a reply to: chr0naut
You said:
"The 1918 flu spread to nearly 100% of the world population over a three year period. COVID-19 has not spread that far. There were lock-downs, and vaccinations, and isolation of those affected with COVID-19, which were not possible in 1918."
Not that's not true just as most of the claims made.
The flu of 1918 infected an estimated 500 million people out of the population of 1.5 billion.
That's why the infection fatality rate is about 10%. Hence about 1/3 of the population was infected and not the entire population of the planet as you have claimed.
www.cdc.gov...
"It is estimated that about 500 million people or one-third of the world’s population became infected with this virus. The number of deaths was estimated to be at least 50 million worldwide with about 675,000 occurring in the United States"
Information provided by the CDC. Note that at least 50 million died which implies that the infection fatality rate could be higher than 10%.
In contrast the IFR of COVID-19 is 0.15% and by February 2021 an estimated 1.5-2 billion people were infected. By October of 2022 you understand that the number of infections may well exceed the 4-5 billion and hence it has infected a much larger percentage of the population in comparison to the Spanish Flu.
pubmed.ncbi.nlm.nih.gov...
"All systematic evaluations of seroprevalence data converge that SARS-CoV-2 infection is widely spread globally. Acknowledging residual uncertainties, the available evidence suggests average global IFR of ~0.15% and ~1.5-2.0 billion infections by February 2021 with substantial differences in IFR and in infection spread across continents, countries and locations"
I think you need to re-evaluate again your claims and arguments as you have clearly confused them. The logic, sequence, and math, don't add up. I understand you are trying to present COVID-19 as the Spanish Flu but anyone with basic high school knowledge can refute your claims.
Before long, Dr. Kathryn Kirkland, an infectious disease specialist at Dartmouth, had a chilling thought: Could she be seeing the start of a whooping cough epidemic?
By late April, other health care workers at the hospital were coughing, and severe, intractable coughing is a whooping cough hallmark.
And if it was whooping cough, the epidemic had to be contained immediately because the disease could be deadly to babies in the hospital and could lead to pneumonia in the frail and vulnerable adult patients there.
It was the start of a bizarre episode at the medical center: the story of the epidemic that wasn’t.
For months, nearly everyone involved thought the medical center had had a huge whooping cough outbreak, with extensive ramifications.
Nearly 1,000 health care workers at the hospital in Lebanon, N.H., were given a preliminary test and furloughed from work until their results were in; 142 people, including Dr. Herndon, were told they appeared to have the disease; and thousands were given antibiotics and a vaccine for protection.
Hospital beds were taken out of commission, including some in intensive care. Then, about eight months later, health care workers were dumbfounded to receive an e-mail message from the hospital administration informing them that the whole thing was a false alarm.
Not a single case of whooping cough was confirmed with the definitive test, growing the bacterium, Bordetella pertussis, in the laboratory. Instead, it appears the health care workers probably were afflicted with ordinary respiratory diseases like the common cold. Now, as they look back on the episode, epidemiologists and infectious disease specialists say the problem was that they placed too much faith in a quick and highly sensitive molecular test that led them astray.
Infectious disease experts say such tests are coming into increasing use and may be the only way to get a quick answer in diagnosing diseases like whooping cough, Legionnaire’s, bird flu, tuberculosis and SARS, and deciding whether an epidemic is under way.
There are no national data on pseudo-epidemics caused by an overreliance on such molecular tests, said Dr. Trish M. Perl, an epidemiologist at Johns Hopkins and past president of the Society of Health Care Epidemiologists of America.
But, she said, pseudo-epidemics happen all the time. The Dartmouth case may have been one the largest, but it was by no means an exception, she said.
“It’s a problem; we know it’s a problem,” Dr. Perl said. “My guess is that what happened at Dartmouth is going to become more common.”
Many of the new molecular tests are quick but technically demanding, and each laboratory may do them in its own way. These tests, called “home brews,” are not commercially available, and there are no good estimates of their error rates.
But their very sensitivity makes false positives likely, and when hundreds or thousands of people are tested, as occurred at Dartmouth, false positives can make it seem like there is an epidemic.
“You’re in a little bit of no man’s land,” with the new molecular tests, said Dr. Mark Perkins, an infectious disease specialist and chief scientific officer at the Foundation for Innovative New Diagnostics, a nonprofit foundation supported by the Bill and Melinda Gates Foundation. “All bets are off on exact performance.”
Of course, that leads to the question of why rely on them at all. “At face value, obviously they shouldn’t be doing it,” Dr. Perl said.
But, she said, often when answers are needed and an organism like the pertussis bacterium is finicky and hard to grow in a laboratory, “you don’t have great options.”
Waiting to see if the bacteria grow can take weeks, but the quick molecular test can be wrong. “It’s almost like you’re trying to pick the least of two evils,” Dr. Perl said.
At Dartmouth the decision was to use a test, P.C.R., for polymerase chain reaction. It is a molecular test that, until recently, was confined to molecular biology laboratories. “That’s kind of what’s happening,” said Dr. Kathryn Edwards, an infectious disease specialist and professor of pediatrics at Vanderbilt University.
“That’s the reality out there. We are trying to figure out how to use methods that have been the purview of bench scientists.”
At Dartmouth, when the first suspect pertussis cases emerged and the P.C.R. test showed pertussis, doctors believed it.
The results seem completely consistent with the patients’ symptoms. “That’s how the whole thing got started,” Dr. Kirkland said. Then the doctors decided to test people who did not have severe coughing. “Because we had cases we thought were pertussis and because we had vulnerable patients at the hospital, we lowered our threshold,” she said.
Anyone who had a cough got a P.C.R. test, and so did anyone with a runny nose who worked with high-risk patients like infants. “That’s how we ended up with 134 suspect cases,” Dr. Kirkland said. And that, she added, was why 1,445 health care workers ended up taking antibiotics and 4,524 health care workers at the hospital, or 72 percent of all the health care workers there, were immunized against whooping cough in a matter of days.
The Dartmouth doctors sent samples from 27 patients they thought had pertussis to the state health departments and the Centers for Disease Control.
There, scientists tried to grow the bacteria, a process that can take weeks. Finally, they had their answer: There was no pertussis in any of the samples.
“We thought, Well, that’s odd,” Dr. Kirkland said. “Maybe it’s the timing of the culturing, maybe it’s a transport problem. Why don’t we try serological testing? Certainly, after a pertussis infection, a person should develop antibodies to the bacteria.”
But when the Centers for Disease Control tested those 39 samples, its scientists reported that only one showed increases in antibody levels indicative of pertussis.
“It was going on for months,” Dr. Kirkland said. But in the end, the conclusion was clear: There was no pertussis epidemic. “We were all somewhat surprised,” Dr. Kirkland said, “and we were left in a very frustrating situation about what to do when the next outbreak comes.”