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originally posted by: tgidkp
originally posted by: MichiganSwampBuck
ETA: I just read your link (Corona Virus Updates Part 3). Holy Christ, maybe they did perfect it and it's ready to wipe us all out.
despite the recent back-patting here on ATS and elsewhere on the web....
... i honestly believe (based on the academic research i have read) that the second "wave" of reinfection is the real killer. we ought not to underestimate the virus' ability to disable the immune system.
again, this is a "gain of function", programmed into the virus, inside a laboratory.
basically, after the body has successfully produced it's antibodies (attached to the immunoglobulin), the virus can come back in and attack the immune system directly by using the antibodies to mediate entry into the cells by modifying the immunoglobulin.... thus disabling the body's immunity to the virus and allowing the viral infection to penetrate more deeply the second time around.
Molecular Mechanism for Antibody-Dependent Enhancement of Coronavirus Entry
what do you think we will be facing
The new viruses will infect many more nearby cells (which can include cells of our immune defence system too, possibly compromising it) and the whole process goes around again, and again, and again.
a reply to: LookingAtMars
It could be that this virus keeps coming back in waves till only the immune are left.
Chinese researchers have raised the possibility that a new subtype pathogen of Covid-19 that has low toxicity but with prolonged ability to infect others might have occurred after observing a rare case in which the disease appeared to be “chronic”, pointing to the possibility of a mutation.
The researchers warn there may be more “chronic infected patients” who carry the infection into their surroundings and trigger an outbreak.
A middle-aged man whose symptoms were not severe appears to have formed a “dynamic balance” with the coronavirus after an extremely prolonged illness lasting 49 days, Chinese military researchers reported in a preprint article on Medrxiv.org last week.
The patient had been observed to have both a high Covid-19 viral load and, at the same time, his immune cell indicators had remained stable.
“The virus and the host may even form a symbiotic relationship,”
originally posted by: tgidkp
a reply to: LookingAtMars
yes, i have read (sorry, no source) that the bugger takes shelter in the nervous system, to become active again when the host is vulnerable.
the article compared it directly to the persistence of the herpes virus breaking out on the mouth.
i admit that i do not know exactly how this will roll out... my only sources are the academics and this is truly a strange bug.
thanks for your contribution.
what do you think we will be facing?
danny.
originally posted by: tgidkp
... i honestly believe (based on the academic research i have read) that the second "wave" of reinfection is the real killer.
we ought not to underestimate the virus' ability to disable the immune system.
. Yikes! That’s scary and it seems to go along with what we’ve heard about people being able to get reinfected and get even more sick than before.
originally posted by: tgidkp
originally posted by: MichiganSwampBuck
ETA: I just read your link (Corona Virus Updates Part 3). Holy Christ, maybe they did perfect it and it's ready to wipe us all out.
despite the recent back-patting here on ATS and elsewhere on the web....
... i honestly believe (based on the academic research i have read) that the second "wave" of reinfection is the real killer. we ought not to underestimate the virus' ability to disable the immune system.
again, this is a "gain of function", programmed into the virus, inside a laboratory.
basically, after the body has successfully produced it's antibodies (attached to the immunoglobulin), the virus can come back in and attack the immune system directly by using the antibodies to mediate entry into the cells by modifying the immunoglobulin.... thus disabling the body's immunity to the virus and allowing the viral infection to penetrate more deeply the second time around.
Molecular Mechanism for Antibody-Dependent Enhancement of Coronavirus Entry