posted on Nov, 4 2018 @ 12:06 AM
Microvascular damage begins to occur at blood glucose levels of 140. T1D & T2D both have a loss of the first-phase insulin response, and this plays a
key role in suppressing hepatic glucose output, which prevents excessive post-prandial glucose excursions, and first-phase kinetics are critical to
preventing postprandial hyperglycemia.
Diabetes is the dysregulation of glucose homeostasis. Diabetes-related complications can be traced back to the deletrious health effects caused by
chronic hyperglycemia. Restoring post-prandial glucose homeostasis is key in the person with T2D.