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originally posted by: Phantom423
a reply to: ServantOfTheLamb
It's not your place to comment on science. You're not a scientist. You're a fraud.
Your opinion is meaningless and ill-conceived. In short, you don't count.
End of message.
originally posted by: ServantOfTheLamb
a reply to: TzarChasm
Glad to see your response was totally relevant to the topic at hand.
If you're not intentionally lying then you have some mental block preventing you from understanding basic biology. Why don't face the fact that you're no good at this. Novel phenotypes are gains in function.
originally posted by: Phantom423
a reply to: chr0naut
Those are good questions. You should look up the answers because they're all in the literature.
Use Google Scholar.
scholar.google.com...
originally posted by: ServantOfTheLamb
a reply to: TzarChasm
resist all you want, but posting poorly formulated attacks on a conspiracy forum is probably not the most effective approach.
....oh the irony...
originally posted by: whereislogic
originally posted by: ServantOfTheLamb
a reply to: TzarChasm
resist all you want, but posting poorly formulated attacks on a conspiracy forum is probably not the most effective approach.
....oh the irony...
So at which point do you reckon a certain kind of people can quit it with the ad hominem-game and get back to addressing anything detailed about the subject that was raised rather than talking about the person who raised it? (other than, oh it's no issue, not interesting, another topic please...along with the 'you just don't understand' and 'you're not a scientist' commentary)?
As long as it works so well...I'm not keeping my hopes up.
Propaganda....it works, bi...you lovers of knowledge that have forgotten* to use their thinking abilities properly and healthily and are capable of so much more than this. (*: not exclusively by their own doing but by bad associations and conditioning with propaganda, while projecting that on those who have been trying to warn you and explain in more detail how it works and how one is affected, as well as the contagious nature of it)
originally posted by: ServantOfTheLamb
a reply to: Phantom423
This illustrates redundancy and it is not what I am talking about.
What do many scientists claim? All living cells fall into two major categories—those with a nucleus and those without. Human, animal, and plant cells have a nucleus. Bacterial cells do not. Cells with a nucleus are called eukaryotic. Those without a nucleus are known as prokaryotic. Since prokaryotic cells are relatively less complex than eukaryotic cells, many believe that animal and plant cells must have evolved from bacterial cells. In fact, many teach that for millions of years, some “simple” prokaryotic cells swallowed other cells but did not digest them. Instead, the theory goes, unintelligent “nature” figured out a way not only to make radical changes in the function of the ingested cells but also to keep the adapted cells inside of the “host” cell when it replicated.9*
*: No experimental evidence exists to show that such an event is possible.
9: 9. Encyclopædia Britannica, CD 2003, “Cell,” “The Mitochondrion and the Chloroplast,” subhead, “The Endosymbiont Hypothesis.”
What qualifies a theory as a scientific theory? According to the Encyclopedia of Scientific Principles, Laws, and Theories, a scientific theory, such as Albert Einstein’s theory of gravity, must
- Be observable
- Be reproducible by controlled experiments
- Make accurate predictions
The same encyclopedia defines a hypothesis as “a more tentative observation of facts [than a theory],” yet lends itself “to deductions that can be experimentally tested.”
"Publish or perish" is a phrase coined to describe the pressure in academia to rapidly and continually publish academic work to sustain or further one's career.
...[whereislogic: now for a description of what I described as "a problem in the so-called scientific community"]
The pressure to publish has been cited as a cause of poor work being submitted to academic journals.
...
This phenomenon has been strongly criticized, the most notable grounds being that the emphasis on publishing may decrease the value of resulting scholarship, as scholars must spend more time scrambling to publish whatever they can get into print, rather than spending time developing significant research agendas.[11] Similarly, humanities scholar Camille Paglia has described the publish or perish paradigm as "tyranny" and further writes that "The [academic] profession has become obsessed with quantity rather than quality. [...] One brilliant article should outweigh one mediocre book."
...
Also, publish-or-perish is linked to scientific misconduct or at least questionable ethics.
...
Genetecist Craig Venter (of the Human Genome Project among other things) in a science forum at Arizona State University, in April 2011, with other scientists discussing evolution, made the comment that there was not likely to be a single tree of life, but rather - many bushes.
Other panelists - like the journalist and writer Richard Dawkins, suggested that Venter, the preeminent genetecist in the world, was wrong, primarily because they hold to the paradigm of a single "tree of life".
(*: note that there is also a more general use of the term endosymbiosis that I'm not referring to, see below what I'm referring to specifically as the mythology surrounding it, so not the general definition that shows up if you google it, some people like to conflate fact with fiction by being vague about details and differences between concepts and notions, see where the bolded parts below for example differ from the general definition; some people also like others to ignore or overlook the logical requirements for their storyline that can also be found in between the bolded parts)
Your source pulled a conclusion out of a hat and just like magic, it looked good, therefore, it worked - who would ever challenge such an innovative conclusion?!! No objective analysis, [irrelevant] lab work, [laughably biased] corroborating references [that are based on the same methodology of making stuff up without proper evidence or even an attempt to prove the claims conclusively, only supported by twisting and cherry-picking facts to paint a picture of what 'may be', or support it, and then sometimes even talking about it as if it's already confirmed to 'be so'] - just a few pages of unreliable [sophistry laced with beguiling technical jargon] with no [proper, reasonable] evidence [, untwisted, not misrepresented or leaving out inconvenient facts that show otherwise or something else, or why the proposed or so-called "evidence" doesn't work logically or reasonably]. The usual scenario which suits their agenda.
The fact that you don't or can't understand studies is not my problem. There are plenty of experiments that show mutations create new alleles. Here are a few, free of charge!
Neo-Darwinian evolution is uniformitarian in that it assumes that all process works the same way, so that evolution of enzymes or flower colors can be used as current proxies for study of evolution of the body plan. It erroneously assumes that change in protein coding sequence is the basic cause of change in developmental program; and it erroneously assumes that evolutionary change in body plan morphology occurs by a continuous process. All of these assumptions are basically counterfactual. This cannot be surprising, since the Neo-Darwinian Synthesis from which these ideas stem was a pre-molecular biology concoction focused on population genetics and adaptation natural history, neither of which have any direct mechanistic import for the genomic regulatory systems that drive embryonic development of the body plan...
Therefore the action of selection differs across dGRN structure. Selection does not operate to produce continuous adaptive change except at the dGRN periphery. The lack of continuous variation in morphogenetic traits defining Class and Phylum level clades is obvious in the striking evolutionary stasis revealed by the fossil record (Davidson and Erwin, 2006; 2009; Erwin, 2011). In other words, while cis-regulatory sequence variation may have continuing adaptive significance at the dGRN periphery, at upper levels of the dGRN hierarchy it does not have the same significance because the system level output is very impervious to change, except for catastrophic loss of the body part or loss of viability altogether.
Bacteria/Microbes Here they identified 66 mutations to that one focal gene which had an adaptive, or beneficial, effect on the fitness of individual
Here they used these results to develop a mathematical model of the probability of a beneficial mutation reaching fixation.
Abstract Lipoprotein lipase (LPL) hydrolyzes triglycerides in the circulation and promotes the hepatic uptake of remnant lipoproteins. Since the gene was cloned in 1989, more than 100 LPL gene mutations have been identified, the majority of which cause loss of enzymatic function. In contrast to this, the naturally occurring LPLS447X variant is associated with increased lipolytic function and an anti-atherogenic lipid profile and can therefore be regarded as a gain-of-function mutation.