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Abstract
Myocarditis and pericarditis are potential post-acute cardiac sequelae of COVID-19 infection, arising from adaptive immune responses. We aimed to study the incidence of post-acute COVID-19 myocarditis and pericarditis. Retrospective cohort study of 196,992 adults after COVID-19 infection in Clalit Health Services members in Israel between March 2020 and January 2021. Inpatient myocarditis and pericarditis diagnoses were retrieved from day 10 after positive PCR. Follow-up was censored on 28 February 2021, with minimum observation of 18 days. The control cohort of 590,976 adults with at least one negative PCR and no positive PCR were age- and sex-matched. Since the Israeli vaccination program was initiated on 20 December 2020, the time-period matching of the control cohort was calculated backward from 15 December 2020. Nine post-COVID-19 patients developed myocarditis (0.0046%), and eleven patients were diagnosed with pericarditis (0.0056%). In the control cohort, 27 patients had myocarditis (0.0046%) and 52 had pericarditis (0.0088%). Age (adjusted hazard ratio [aHR] 0.96, 95% confidence interval [CI]; 0.93 to 1.00) and male sex (aHR 4.42; 95% CI, 1.64 to 11.96) were associated with myocarditis. Male sex (aHR 1.93; 95% CI 1.09 to 3.41) and peripheral vascular disease (aHR 4.20; 95% CI 1.50 to 11.72) were associated with pericarditis. Post COVID-19 infection was not associated with either myocarditis (aHR 1.08; 95% CI 0.45 to 2.56) or pericarditis (aHR 0.53; 95% CI 0.25 to 1.13). We did not observe an increased incidence of neither pericarditis nor myocarditis in adult patients recovering from COVID-19 infection.
5. Conclusions
Our data suggest that there is no increase in the incidence of myocarditis and pericarditis in COVID-19 recovered patients compared to uninfected matched controls. Further longer-term studies will be needed to estimate the incidence of pericarditis and myocarditis in patients diagnosed with COVID-19.
originally posted by: Antisocialist
a reply to: LordAhriman
Isn't this discussion fairly irrelevant, considering where the virus originated and for what purpose it was originated? So one gets myocarditis or pericarditis from the virus or from the vaccine is irrelevant considering that the original intent of both is to reduce global populations.
Damned if you do, damned if you don't.
originally posted by: Kenzo
No link between COVID-19 the disease in an unvaccinated population and either myocarditis or pericarditis. Over 200,000 people in the study group. This important to know yes ? because we are talking so much about the sudden deaths/Myocarditis .
The Incidence of Myocarditis and Pericarditis in Post COVID-19 Unvaccinated Patients—A Large Population-Based Study
Abstract
Myocarditis and pericarditis are potential post-acute cardiac sequelae of COVID-19 infection, arising from adaptive immune responses. We aimed to study the incidence of post-acute COVID-19 myocarditis and pericarditis. Retrospective cohort study of 196,992 adults after COVID-19 infection in Clalit Health Services members in Israel between March 2020 and January 2021. Inpatient myocarditis and pericarditis diagnoses were retrieved from day 10 after positive PCR. Follow-up was censored on 28 February 2021, with minimum observation of 18 days. The control cohort of 590,976 adults with at least one negative PCR and no positive PCR were age- and sex-matched. Since the Israeli vaccination program was initiated on 20 December 2020, the time-period matching of the control cohort was calculated backward from 15 December 2020. Nine post-COVID-19 patients developed myocarditis (0.0046%), and eleven patients were diagnosed with pericarditis (0.0056%). In the control cohort, 27 patients had myocarditis (0.0046%) and 52 had pericarditis (0.0088%). Age (adjusted hazard ratio [aHR] 0.96, 95% confidence interval [CI]; 0.93 to 1.00) and male sex (aHR 4.42; 95% CI, 1.64 to 11.96) were associated with myocarditis. Male sex (aHR 1.93; 95% CI 1.09 to 3.41) and peripheral vascular disease (aHR 4.20; 95% CI 1.50 to 11.72) were associated with pericarditis. Post COVID-19 infection was not associated with either myocarditis (aHR 1.08; 95% CI 0.45 to 2.56) or pericarditis (aHR 0.53; 95% CI 0.25 to 1.13). We did not observe an increased incidence of neither pericarditis nor myocarditis in adult patients recovering from COVID-19 infection.
5. Conclusions
Our data suggest that there is no increase in the incidence of myocarditis and pericarditis in COVID-19 recovered patients compared to uninfected matched controls. Further longer-term studies will be needed to estimate the incidence of pericarditis and myocarditis in patients diagnosed with COVID-19.
originally posted by: Kenzo
I just put this here also :
compilation: peer reviewed medical papers of covid vaccine injuries
originally posted by: chr0naut
originally posted by: Antisocialist
a reply to: LordAhriman
Isn't this discussion fairly irrelevant, considering where the virus originated and for what purpose it was originated? So one gets myocarditis or pericarditis from the virus or from the vaccine is irrelevant considering that the original intent of both is to reduce global populations.
Damned if you do, damned if you don't.
If the intention was to depopulate, why did they use such a futile method?
Surely they could have faked a world war and deployed nukes, and/or have used really deadly pathogens or poisons?
originally posted by: Kenzo
Agree, they do anything to try hide the truth .
People will loose trust to whole medical science , because the jabs but also all other cover up`s etc..at least part of the population, Covidians will take 10th booster if they are still here then..
originally posted by: Kenzo
a reply to: LordAhriman
The OP study was done with unvaccinated people.
The study you speak was done with vaccinated people
originally posted by: chr0naut
originally posted by: Antisocialist
a reply to: LordAhriman
Isn't this discussion fairly irrelevant, considering where the virus originated and for what purpose it was originated? So one gets myocarditis or pericarditis from the virus or from the vaccine is irrelevant considering that the original intent of both is to reduce global populations.
Damned if you do, damned if you don't.
If the intention was to depopulate, why did they use such a futile method?
Surely they could have faked a world war and deployed nukes, and/or have used really deadly pathogens or poisons?