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originally posted by: Rich Z
Well, I am pushing the envelope a LOT in my understanding how this all works, but I would think that if a virus is using a particular spike protein to effect an attachment to a target cell via it's ACE2 receptor, aren't those target cell receptors already primed to receive attachments from some beneficial cells by design? In other words, aren't those receptors on those cells for a needful reason?
This conclusion suggests that vaccination-generated antibody and/or exogenous antibody against S protein not only protects the host from SARS-CoV-2 infectivity but also inhibits S protein-imposed endothelial injury.
originally posted by: dragonridr
a reply to: ElectricUniverse
You know what's funny? You got it completely backwards the study is suggesting the vaccine helps protect you from damage by the corona virus.
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