Link Covid-19 actively targets T cells

This is disturbing
If this is part of how this virus works it explains the high mortality rate

www.scmp.com...

www.ncbi.nlm.nih.gov...

a reply to: flashbackjack

So, if those wet markets do not close effing NOW, we do not need to give a name to the next version of that bug. We'll be done way before.

Helps support the idea that CORVOD-19 was engineered with segments from HIV. That first link claims that CORVID-19 has all the worst attributes of all the worst viruses, supporting that it is a manufactured viral chimera. The best bio-weapons have a high mortality rate and no cure or vaccine.

a reply to: flashbackjack

I find this part of the first article extremely interesting:

But some deadly human viruses such as Aids and Ebola have similar sequences, prompting speculation that the novel coronavirus might have been spreading quietly in human societies for a long time before causing this pandemic.

A whole lot of people suspect that they may have already had this virus last year, myself and my husband included, and tested negative for flu and pneumonia. We were sick for weeks and the dry cough lingered for a few months after. The doctor's best guess as to what it could be was Human Para-influenza (basically Kennel Cough for humans). Since this virus first hit the news we have been labeled as crazy and told that the virus didn't exist here then. Perhaps the experts were wrong.

originally posted by: deckdel
a reply to: flashbackjack

So, if those wet markets do not close effing NOW, we do not need to give a name to the next version of that bug. We'll be done way before.

I would be more worried about the bio-labs that have constant leaks. The so called "bats" this came from are not at the wet market but coincidentally the Wuhan lab just happened to go get those bats just before the outbreak.

www.dailymail.co.uk...

The lab is the center of several conspiracy theories that suggest it is the original source of the coronavirus outbreak

The institute experimented on bats from the source of the coronavirus

They were captured more than 1,000 miles away in Yunnan

Sequencing of the Covid-19 genome has traced it to bats to Yunnan's caves

a reply to: infolurker



Long video. But, everything you need to know ... takes a while to take in.

Zomg T virus confirmed zombie apocalypse is totally real

originally posted by: deckdel
a reply to: flashbackjack

So, if those wet markets do not close effing NOW, we do not need to give a name to the next version of that bug. We'll be done way before.

What about the bird flu and the swine flu? Same things.
We should close all the chicken farms and pig farms also...But NO because that would hurt america.

originally posted by: Centsay

originally posted by: deckdel
a reply to: flashbackjack

So, if those wet markets do not close effing NOW, we do not need to give a name to the next version of that bug. We'll be done way before.

What about the bird flu and the swine flu? Same things.
We should close all the chicken farms and pig farms also...But NO because that would hurt america.

Shouldent it be the wild game markets in america I guess that would require you to know what your talking about first

But I seriously doubt it came from the wet markets normal operation it sounds more like they sold test animals to the market

Dose the virus strain they found in the wild also attack t cells or is that the other 4% that dosent match I really wonder if the virus came from the wild and they nurtured/gmo it to attack the immune system so it's normal symtoms are much worse

Probly something we will never know

originally posted by: flashbackjack
This is disturbing
If this is part of how this virus works it explains the high mortality rate

www.scmp.com...

www.ncbi.nlm.nih.gov...

As in Resident Evil T-Virus Zombies ??

Just kidding, Yea if it targets our T-Cells that is bad bad news for our immune systems, Possibly even means we will not build a natural Immunity which is very scary.

What If a protein variant between Covid19/HIV has been incubated in all of us as a sleeper protein/virus that will be activated by a process unknown.

Then Covid/Corona that is a mutational virus of 5 or maybe 6 base origins.

So HIV is a retro virus that can not be cured only made undetectable in the blood basically a person's Viral load ??

Do we then, have to urgently find inhibitors, for both Corona mutations and HIV, its strange that HIV treatment is sometimes called combination therapy or ART Antiretroviral therapy.

I wonder was this trailed in Africa? and then amongst other communities in the last 30 years or so before being refined and released on us all ????

Keep an eye out for references to Karposis Sarcoma in relation to covid ???


Ranitidine keeps on popping up WHY

A 24-year-old, Chinese HIV-infected patient with a 2-year treatment history with tenofovir, lamivudine and efavirenz (CD4 cell count and HIV-RNA levels unreported), had a non-severe course of COVID-19 Lopinavir/ritonavir had been added to the antiretroviral regimen after COVID-19 diagnosis.

www.ncbi.nlm.nih.gov...

A further Chinese patient living with HIV had 34 CD4 cells/μL and a prolonged course of COVID-19 (12). An additional HIV-infected patient with fever, muscle aches and right lower lobe pneumonia at a chest CT scan was reported by Chinese authors from Shenzhen (13). However, SARS-CoV-2 RNA was persistently negative on different specimen samples at various times during the course of his illness (13), and we cannot therefore be sure that this patient was SARS-CoV-2-coinfected.
Three HIV-coinfected cases have been reported from Italy (16). A 62-year-old man with undetectable viral load and 441 CD4 cells/μL required mechanical ventilation and improved; a 63-year-old man with undetectable HIV-RNA and 743 CD4 cells/μL and a 57-year-old woman (HIV-RNA and CD4 cell count not reported) had an uneventful course (16). Interestingly, prior to getting SARS-CoV-2 all the three patients were on darunavir-based antiretroviral therapy, and pharmacokinetic data showed good compliance, suggesting that darunavir, at least at the currently employed 800 mg dosage, does not prevent SARS-CoV-2 infection HIV-infected individuals (16). It must be stressed that Janssen reported on March 18, 2020, that darunavir is not effective against SARS-CoV-2 due to low affinity to coronavirus protease.

www.ncbi.nlm.nih.gov...


Okay...
The co-existence of the two epidemics of HIV and SARS-CoV-2 could be particularly deleterious for people living with HIV not only in low and middle-income countries but also in high income countries. Widespread lockdowns, enforced in an attempt to curb the spread of SARS-CoV-2 infection, lead to patients' job losses, difficulties in reaching the clinics where anti-HIV drugs are distributed, and problems in drug supplies to the clinics. Funds needed to step up the response to the new pandemic could reduce those assigned to the fight against HIV infection/AIDS and TB, and vulnerable HIV-infected populations (drug users, sex workers, poor patients living in urban slums or in rural areas, prisoners) would particularly suffer from this. Any efforts will have to be made to prevent or limit the above problems. In any case, it will be extremely important to describe the features of SARS-CoV-2 infection and the evolution of COVID-19 in HIV-infected patients, including whether HIV-infected people develop sufficient level of anti-SARS-CoV-2 antibodies, and their persistence over time. Even in this difficult situation, clinical and research centres, including those in Africa, will have to strive to clarify the numerous aspects of this unprecedented coinfection for the benefit of all HIV-infected patients worldwide.

www.ncbi.nlm.nih.gov...

Pharmacology letter Accelerated communication
The effect of histamine type 2 receptor antagonists on human immunodeficiency virus (HIV) replication: Identification of a new class of antiviral agents
Author links open overlay panelAldar S.BourinbaiarEric C.Fruhstorfer
doi.org...(96)00553-XGet rights and content
Abstract
The suppression of human immunodeficiency virus (HIV) replication by histamine type 2 (H2) receptor antagonists, cimetidine, ranitidine, and famotidine was examined in vitro. The 50% reduction (IC50) in p24 antigen expression caused by the anti-ulcer agent, cimetidine, was observed at 26.8 nM with IC100 at 10 μM. Unlike azidothymidine — a reverse transcriptase inhibitor — cimetidine blocked HIV infection without affecting cell growth, as no cytotoxicity was observed even at the highest 1 mM dose. Ranitidine and famotidine were less potent than cimetidine. H1 antagonists, cyproheptadine and diphenhydramine, had no effect on HIV replication. Although the activity of cimetidine was observed at concentrations attainable by oral administration, the mechanism of anti-HIV action is unknown. This is the first report on antiretroviral action of H2 blockers. Further studies are warranted to establish the potential of a new class of anti-HIV agents with immunomodulating properties.

pubmed.ncbi.nlm.nih.gov...

Ranitidine keeps on popping up WHY

In UK Ranitidine was pulled in early November LITERALLY OVERNIGHT ?? Stomach Alkaline immune system




I could be a Genius, mentally ill, Drunk, or ...on Drugs even all four

a reply to: Fingle

I think its a done deal............ already, as now in UK office of national statistics is paying people £50 for first visit and then £25 per person to take bloods for Covid tests FOR A YEAR FACT erm Data/DNA harvesting................