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originally posted by: flanimal4114
This is getting very personal, so I'm gonna leave this for u guys bye
Ok, so by "information" you do not mean the genetic code.
originally posted by: ServantOfTheLamb
Yes and No. The information in DNA is tied to the genetic code just like the information in a book is tide to the words. Also i already told you that I think IC applies to entire systems rather than just one select point. As DNA on its own is pretty worthless
originally posted by: flanimal4114
a reply to: GetHyped
originally posted by: flanimal4114
This is getting very personal, so I'm gonna leave this for u guys bye
i men't against the others, not me, im fine, i didnt want bracs or the others getting, well, overboard. ill come back happily.
originally posted by: TheLamb
Explain then why deer are increasingly eating birds. Evolution has all the answers, doesn't it? Haven't deer evolved to eat plants? Should't that provide it with the nutrients it requires? Deer shouldn't be able to process meat according to Evolution after millions of years as herbivores. And how have they suddenly acquired the skill to hunt a flying prey? Shouldn't that have evolved?
So which part or system is irreducibly complex
Some people say that flagellum applies, some folks argue that the eye is IC, but both can be explained by incremental changes.
Information does not apply since it is human data that comes from genetic code
Abstract
Four decades ago, several scientists suggested that the impossibility of any evolutionary process sampling anything
but a miniscule fraction of the possible protein sequences posed a problem for the evolution of new proteins. This
potential problem—the sampling problem—was largely ignored, in part because those who raised it had to rely on
guesswork to fill some key gaps in their understanding of proteins. The huge advances since that time call for a careful
reassessment of the issue they raised. Focusing specifically on the origin of new protein folds, I argue here that
the sampling problem remains. The difficulty stems from the fact that new protein functions, when analyzed at the
level of new beneficial phenotypes, typically require multiple new protein folds, which in turn require long stretches
of new protein sequence. Two conceivable ways for this not to pose an insurmountable barrier to Darwinian searches
exist. One is that protein function might generally be largely indifferent to protein sequence. The other is that relatively
simple manipulations of existing genes, such as shuffling of genetic modules, might be able to produce the
necessary new folds. I argue that these ideas now stand at odds both with known principles of protein structure and
with direct experimental evidence. If this is correct, the sampling problem is here to stay, and we should be looking
well outside the Darwinian framework for an adequate explanation of fold origins.
Since there are
n^ℓ
possible ways to construct a polymer chain of length ℓ from n
distinct monomer types, amino acid chains a mere twelve residues
long (residue being the term for an amino acid monomer that has
been incorporated into a protein polymer) can be built in four quadrillion
ways (20^12 = 4 × 10^15). The gene sequences that encode
these short chains are even more numerous as a consequence of
the many-to-one mapping of codons (the n
The simple relationship between gene sequences and protein
sequences in bacteria allows protein sizes to be determined directly
from genomic data. This, in combination with abundant
data on protein structures and functions, makes the well studied
gut bacterium Escherichia coli an excellent model system
for examining a simple proteome.
The size of E. coli proteins with known functions can be assessed by analyzing the data files
provided by EcoCyc , a comprehensive database for this organism.
Figure 1 shows the distribution of protein chain lengths
for all proteins known to be involved in enzymatic functions in
E. coli, either alone or in combination with other proteins. From
the mode of the distribution we see that the most common length
of these proteins is around 300 amino acid residues, with the
higher mean and median lengths reflecting the existence of numerous
protein chains that are much longer than this.
If we take 300 residues as a typical chain length for functional
proteins, then the corresponding set of amino acid sequence possibilities
is unimaginably large, having 20^300 ( = 10^390) members.
originally posted by: ServantOfTheLamb
The cell is irreducibly complex. A cell membrane on its own cannot replicate, and as such is not alive . It on its own cannot work and without it you have no cell structure for organelles and cytoplasm and all that fun stuff in the membrane isn't container so no function cell. Any organelle including the mitochondria removed is worthless on its own. The mitochondria's structure and functioning is governed by the nucleus and they cannot exist in a cell-free culture nor can they reproduce without nuclear participation. Then you have a ribosome. Remove this you have no proteins. A ribosome in and of itself is an incredibly complex structure.
Irreducible complexity (IC) is a pseudoscientific argument that postulates that certain biological systems are too complex to have evolved from simpler or "less complete" predecessors through natural selection acting upon a series of advantageous naturally occurring chance mutations.
Well your talking about a science not a philosophy. Show me evidence that these supposed incremental changes can occur, but as of right now I would disagree with this statement.
How exactly is the information for building a protein human information? A protein functions based on its shape. Its shape is determined by the sequence of amino acids which is determined by the sequence of nucleotide bases in the mRNA template of DNA. Then somewhere hidden in there is also the information for the correct protein structure or fold.
Can you describe and present evidence for the mechanism that stops genetic differences in populations from accumulating to the point of speciation?
originally posted by: wisvol
To answer the title question:
Genetic differences accumulate endlessly.
Speciation is another bowl of wax, and isn't a point to be reached by accumulated genetic difference.
Mendel's genetics shows this so elegantly, Darwin had to take it from meaning "peas that taste better or resist mold better do so because they're coded differently through their generations" into "peas become mangoes if you wait longer than possible to wait", and that was an error still repeated today for its psychosocial implications.
So the mechanism would be there isn't one: genetic difference isn't speciation.
Genetic difference is evolution. Speciation is not a separate process. The op is in reference to the incorrect assertion from the creationists claiming micro evolution is somehow a separate process from macro evolution (as opposed to taxonomic terminology). In order for that claim to be accurate, there must be an identifiable mechanism.
Regardless of how a person tries to spin, at the end of the day, all life on earth is defined by the genetic makeup. The building blocks are the same, the only defing difference is the amout and order of those blocks.
The mechanisms that change the genetic code are very well understood, far beyond Darwin and Mendel. Those changes are what we use as a classification system.
originally posted by: wisvol
a reply to: Cypress
Genetic difference is actual evolution, and the semantically drifted term "evolution" referring to the origin of species as other species is an incorrect notion.
Speciation is a distinct process that despite claims to the contrary isn't observed, because of the unexperimentable amount of time required for theoretical speciaton.
Micro and Macro don't apply to genetic difference. Macro genetic difference such as an extra chromosome is either lethal or gives birth to severely impaired individuals.
The barren nature of the ligers you mention is a hint as to the very narrow nature of genetic compatibility.
If you need a mechanism as phrased by the OP, name the mechanism by which small changes in temperature according to seasons do not accumulate to below zero Kelvin or above sun temperature.
Same here
Offspring are different from their parents in ways not inclusive of their species.
Speciation is a failed theory, as demonstrated through several attempts to speciate creatures with a short generational period such as drosophilia, who decades after being separated from their main population are still interfertile although they may be irradiated and maybe albino.
Regardless of how a person tries to spin, at the end of the day, all life on earth is defined by the genetic makeup. The building blocks are the same, the only defing difference is the amout and order of those blocks.
Indeed, now replace "genetic" with "elemental", as all life if made mostly of coal and water.
Those building blocks are the same, and leaving a lump of coal in a glass of water for a million years will not yield fish any more than leaving fish in a pond for a billion years yield another species.
The mechanisms that change the genetic code are very well understood, far beyond Darwin and Mendel. Those changes are what we use as a classification system.
The term Evolution has not "semantically drifted", in terms of biological evolution, it is a specificly defined process. To ignore and disregard the proper use of the definition would be ignorance as is evidenced in your post.
Speciation is not a process. If it was it would have a specific mechanism. It does not. There is only evolution. It's not even technically a true term because there is no process of speciation.
Actually, ligers are not barren. Only under extreme conditions would species interbreed if a close enough relation exists but it is possible.
First of all you do not understand what the OP is asking. I will repeat from my last post; The Op is addressing the issue brought up by creationist claims stating that adaptation occurs in populations but that there is no divergence in populations. They separate evolution into 2 separate processes. In order to do this there must be a mechanism that prevents or would be required to get a divergence. The OP was asking for for identification of that mechanism and evidence of supporting that claim
Speciation is not a theory because there is no process of speciation. There is only evolution. The term species is only a classification term for organizing relationships. Speciation is a slang term used to denote an approximate range of when we see two species diverge from a common ancestor. As for genetic differences, you claim they do not leave their parent species yet we share a majority of our DNA with all life on earth and the changes in DNA can be directly correlated with known processes in genetics. So your claim is bogus with all the evidence showing the contrary.
While the atoms may largely be the same, the chemical processes involved in biochemistry =/= mixing coal in water. It's not an appropriate chemical reaction and is an absurd claim. This is a straw man.
"And still, a creature as simple and rapidly breeding as drosophilia cannot be observed to produce another species because the origin of species, despite popular belief, is not other species. "
Life has been on Earth for upwards of 3 billion years and you expect us to watch single celled organisms morph into multicellular organisms in just a few years.