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Fever was the most common symptom reported by patients with 80 per cent suffering a raised temperature,
originally posted by: MrLimpet
a reply to: infolurker
Key findings from the article notes.
Fever was the most common symptom reported by patients with 80 per cent suffering a raised temperature,
Meaning 20% don't have a fever, but have the virus.
Quite concerning since a fever (seems to be what they are saying) determines if you have it or not.
originally posted by: MysterX
This is why Spanish Flu killed mostly younger people, also why Swine Flu took a harder toll on younger people...their immune systems were stronger, which provided a stronger immune response to the viral infection, creating all the conditions for their own bodies to kill them.
Why then i wonder, should Ebola be any different?
originally posted by: Aleister
This just goes to show that older folks really have to change many of their lifelong habits, and gain new ones, to strengthen their immune systems.
originally posted by: dianajune
originally posted by: MrLimpet
a reply to: infolurker
Key findings from the article notes.
Fever was the most common symptom reported by patients with 80 per cent suffering a raised temperature,
Meaning 20% don't have a fever, but have the virus.
Quite concerning since a fever (seems to be what they are saying) determines if you have it or not.
I agree this is concerning as someone can be infected and not have any symptoms, inc. fever. This is why quarantining is an absolute necessity.
This is why so many people are upset with the selfish, self-absorbed CDC Nurse Kaci.
This is why so many people are upset with Dr. Nancy Snyderman, who defied her quarantine to get takeaway food.
This is why so many people are upset with Dr. Craig Spencer for having lied to the NYPD about his supposed "quarantine."
It feels like those who are opposed to quarantines WANT Ebola to spread. They make no sense whatever.
In general, when virus infection frenzied the genes involved in promoting blood vessel inflammation and cell death, serious or fatal disease followed. On the other hand, survivors experienced more activity in genes that order blood vessel repair and the production of infection-fighting white blood cells.
The scientists note that certain specialized types of cells in the liver could also have limited virus reproduction and put a damper on systemic inflammation and blood clotting problems in resistant mice. Susceptible mice had widespread liver infection, which may explain why they had more virus in their bodies and poorly regulated blood coagulation. The researchers also noticed that spleens in the resistant and susceptible mice took alternate routes to try to ward off infection.